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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
3
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pubmed:dateCreated |
2009-9-29
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pubmed:abstractText |
Hematopoietic progenitor cells (HPCs) can improve cardiac function after myocardial infarction. However, occurrence of arrhythmias is a potential limitation of cell therapy. In this study, we investigated the cardiac electrophysiological properties of ex vivo expanded HPCs, generated by beta-catenin gene transfer, after transcoronary delivery in a murine model of ischemia/reperfusion (I/R) injury.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:issn |
1421-9751
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pubmed:author |
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pubmed:copyrightInfo |
Copyright 2009 S. Karger AG, Basel.
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pubmed:issnType |
Electronic
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pubmed:volume |
114
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
199-207
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pubmed:meshHeading |
pubmed-meshheading:19602881-Animals,
pubmed-meshheading:19602881-Arrhythmias, Cardiac,
pubmed-meshheading:19602881-Cell Line,
pubmed-meshheading:19602881-Electrocardiography, Ambulatory,
pubmed-meshheading:19602881-Electrophysiologic Techniques, Cardiac,
pubmed-meshheading:19602881-Gene Therapy,
pubmed-meshheading:19602881-Gene Transfer Techniques,
pubmed-meshheading:19602881-Hematopoietic Stem Cell Transplantation,
pubmed-meshheading:19602881-Humans,
pubmed-meshheading:19602881-Male,
pubmed-meshheading:19602881-Mice,
pubmed-meshheading:19602881-Mice, Inbred C57BL,
pubmed-meshheading:19602881-Myocardial Infarction,
pubmed-meshheading:19602881-Myocardial Reperfusion Injury,
pubmed-meshheading:19602881-Myocardium,
pubmed-meshheading:19602881-Telemetry,
pubmed-meshheading:19602881-beta Catenin
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pubmed:year |
2009
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pubmed:articleTitle |
Arrhythmia susceptibility in mice after therapy with beta-catenin-transduced hematopoietic progenitor cells after myocardial ischemia/reperfusion.
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pubmed:affiliation |
Department of Cardiovascular Medicine, Hannover Medical School, Hannover, Germany.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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