pubmed-article:19592464 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:19592464 | lifeskim:mentions | umls-concept:C1135918 | lld:lifeskim |
pubmed-article:19592464 | lifeskim:mentions | umls-concept:C0221464 | lld:lifeskim |
pubmed-article:19592464 | lifeskim:mentions | umls-concept:C0007595 | lld:lifeskim |
pubmed-article:19592464 | lifeskim:mentions | umls-concept:C0020507 | lld:lifeskim |
pubmed-article:19592464 | lifeskim:mentions | umls-concept:C1609943 | lld:lifeskim |
pubmed-article:19592464 | lifeskim:mentions | umls-concept:C1999216 | lld:lifeskim |
pubmed-article:19592464 | pubmed:issue | 9 | lld:pubmed |
pubmed-article:19592464 | pubmed:dateCreated | 2009-8-20 | lld:pubmed |
pubmed-article:19592464 | pubmed:abstractText | Arterial injury induces smooth muscle cell (SMC) proliferation, migration, and intimal accumulation of cells and extracellular matrix. These processes are regulated by the administration of the glycosaminoglycans heparin and heparan sulfate, but little is known about the role of endogenous heparan sulfate proteoglycans in the vessel wall. We investigated the response to carotid injury of syndecan-1-null mice to assess the function of one of a conserved family of transmembrane heparan and chondroitin sulfate proteoglycans. | lld:pubmed |
pubmed-article:19592464 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:19592464 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:19592464 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:19592464 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:19592464 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:19592464 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:19592464 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:19592464 | pubmed:language | eng | lld:pubmed |
pubmed-article:19592464 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:19592464 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:19592464 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:19592464 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:19592464 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:19592464 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:19592464 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:19592464 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:19592464 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:19592464 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:19592464 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:19592464 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:19592464 | pubmed:month | Sep | lld:pubmed |
pubmed-article:19592464 | pubmed:issn | 1524-4636 | lld:pubmed |
pubmed-article:19592464 | pubmed:author | pubmed-author:KenagyRichard... | lld:pubmed |
pubmed-article:19592464 | pubmed:author | pubmed-author:ClowesAlexand... | lld:pubmed |
pubmed-article:19592464 | pubmed:author | pubmed-author:ChenLihuaL | lld:pubmed |
pubmed-article:19592464 | pubmed:author | pubmed-author:FukaiNozomiN | lld:pubmed |
pubmed-article:19592464 | pubmed:author | pubmed-author:GaoLuL | lld:pubmed |
pubmed-article:19592464 | pubmed:author | pubmed-author:DaumGuenterG | lld:pubmed |
pubmed-article:19592464 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:19592464 | pubmed:volume | 29 | lld:pubmed |
pubmed-article:19592464 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:19592464 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:19592464 | pubmed:pagination | 1356-62 | lld:pubmed |
pubmed-article:19592464 | pubmed:dateRevised | 2010-9-7 | lld:pubmed |
pubmed-article:19592464 | pubmed:meshHeading | pubmed-meshheading:19592464... | lld:pubmed |
pubmed-article:19592464 | pubmed:meshHeading | pubmed-meshheading:19592464... | lld:pubmed |
pubmed-article:19592464 | pubmed:meshHeading | pubmed-meshheading:19592464... | lld:pubmed |
pubmed-article:19592464 | pubmed:meshHeading | pubmed-meshheading:19592464... | lld:pubmed |
pubmed-article:19592464 | pubmed:meshHeading | pubmed-meshheading:19592464... | lld:pubmed |
pubmed-article:19592464 | pubmed:meshHeading | pubmed-meshheading:19592464... | lld:pubmed |
pubmed-article:19592464 | pubmed:meshHeading | pubmed-meshheading:19592464... | lld:pubmed |
pubmed-article:19592464 | pubmed:meshHeading | pubmed-meshheading:19592464... | lld:pubmed |
pubmed-article:19592464 | pubmed:meshHeading | pubmed-meshheading:19592464... | lld:pubmed |
pubmed-article:19592464 | pubmed:meshHeading | pubmed-meshheading:19592464... | lld:pubmed |
pubmed-article:19592464 | pubmed:meshHeading | pubmed-meshheading:19592464... | lld:pubmed |
pubmed-article:19592464 | pubmed:meshHeading | pubmed-meshheading:19592464... | lld:pubmed |
pubmed-article:19592464 | pubmed:meshHeading | pubmed-meshheading:19592464... | lld:pubmed |
pubmed-article:19592464 | pubmed:meshHeading | pubmed-meshheading:19592464... | lld:pubmed |
pubmed-article:19592464 | pubmed:meshHeading | pubmed-meshheading:19592464... | lld:pubmed |
pubmed-article:19592464 | pubmed:meshHeading | pubmed-meshheading:19592464... | lld:pubmed |
pubmed-article:19592464 | pubmed:meshHeading | pubmed-meshheading:19592464... | lld:pubmed |
pubmed-article:19592464 | pubmed:meshHeading | pubmed-meshheading:19592464... | lld:pubmed |
pubmed-article:19592464 | pubmed:meshHeading | pubmed-meshheading:19592464... | lld:pubmed |
pubmed-article:19592464 | pubmed:meshHeading | pubmed-meshheading:19592464... | lld:pubmed |
pubmed-article:19592464 | pubmed:meshHeading | pubmed-meshheading:19592464... | lld:pubmed |
pubmed-article:19592464 | pubmed:meshHeading | pubmed-meshheading:19592464... | lld:pubmed |
pubmed-article:19592464 | pubmed:meshHeading | pubmed-meshheading:19592464... | lld:pubmed |
pubmed-article:19592464 | pubmed:year | 2009 | lld:pubmed |
pubmed-article:19592464 | pubmed:articleTitle | Syndecan-1: an inhibitor of arterial smooth muscle cell growth and intimal hyperplasia. | lld:pubmed |
pubmed-article:19592464 | pubmed:affiliation | Department of Surgery and Center for Cardiovascular Biology, University of Washington, Seattle, WA 98195-6410, USA. | lld:pubmed |
pubmed-article:19592464 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:19592464 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
pubmed-article:19592464 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
entrez-gene:20969 | entrezgene:pubmed | pubmed-article:19592464 | lld:entrezgene |
http://linkedlifedata.com/r... | entrezgene:pubmed | pubmed-article:19592464 | lld:entrezgene |