Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
1
pubmed:dateCreated
2010-6-14
pubmed:abstractText
Interleukin (IL)-17F is involved in lung inflammation, but the effect of IL-17F on endothelial permeability and its signaling pathway remain ill-defined. The current study sought to investigate the effect of IL-17F on endothelium and assess the role of protein kinase C (PKC) and src-suppressed C kinase substrate (SSeCKS) in this process.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Jul
pubmed:issn
1095-8673
pubmed:author
pubmed:copyrightInfo
(c) 2010. Published by Elsevier Inc. All rights reserved.
pubmed:issnType
Electronic
pubmed:volume
162
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
110-21
pubmed:meshHeading
pubmed:year
2010
pubmed:articleTitle
Interleukin-17F-induced pulmonary microvascular endothelial monolayer hyperpermeability via the protein kinase C pathway.
pubmed:affiliation
Department of Respiratory Medicine, First Affiliated Hospital of Anhui Medical University, Anhui, Hefei, China.
pubmed:publicationType
Journal Article, Research Support, Non-U.S. Gov't