Source:http://linkedlifedata.com/resource/pubmed/id/19481824
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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
2
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pubmed:dateCreated |
2010-12-7
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pubmed:abstractText |
Inflammation is important in plaque vulnerability but the role of atheroprotective mediators in unstable plaques is not defined. The apolipoproteinA-I (apoA-I) component of HDL, and CD4+/CD25+ regulatory T cells (with their major transcription factor, Foxp3), have been implicated in the suppression of vascular inflammation. Our aim was to characterise the presence of these novel "protective" markers (apoA-I and Foxp3) in carotid plaques from symptomatic and asymptomatic subjects.
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pubmed:commentsCorrections | |
pubmed:language |
eng
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pubmed:journal | |
pubmed:citationSubset |
IM
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pubmed:chemical | |
pubmed:status |
MEDLINE
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pubmed:month |
Nov
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pubmed:issn |
1874-1754
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pubmed:author | |
pubmed:copyrightInfo |
Crown Copyright © 2009. Published by Elsevier Ireland Ltd. All rights reserved.
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pubmed:issnType |
Electronic
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pubmed:day |
19
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pubmed:volume |
145
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
183-7
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pubmed:meshHeading |
pubmed-meshheading:19481824-Aged,
pubmed-meshheading:19481824-Apolipoprotein A-I,
pubmed-meshheading:19481824-Atherosclerosis,
pubmed-meshheading:19481824-Carotid Stenosis,
pubmed-meshheading:19481824-Female,
pubmed-meshheading:19481824-Forkhead Transcription Factors,
pubmed-meshheading:19481824-Humans,
pubmed-meshheading:19481824-Male
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pubmed:year |
2010
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pubmed:articleTitle |
The "atheroprotective" mediators apolipoprotein A-I and Foxp3 are over-abundant in unstable carotid plaques.
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pubmed:affiliation |
Department of Cardiology, Royal Prince Alfred Hospital, Sydney, Australia. sanjay.patel@bigpond.com
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pubmed:publicationType |
Journal Article,
Comparative Study,
Research Support, Non-U.S. Gov't
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