Source:http://linkedlifedata.com/resource/pubmed/id/19321208
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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
6
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pubmed:dateCreated |
2009-5-15
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pubmed:abstractText |
To date, major works on tracheomalacia have assumed a structural etiology and have proposed therapies as such. We describe a possible neurologic etiology for tracheomalacia in a child with clinically significant tracheomalacia that resolved in synchrony with each treatment of his recurring hydrocephalus. Endoscopy confirms remarkable expansion of tracheal diameter 7 days after decreasing intracranial pressure. The possibility of a neurologic etiology for tracheomalacia casts this condition in a new light with potential therapeutic implications.
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pubmed:language |
eng
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pubmed:journal | |
pubmed:citationSubset |
IM
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pubmed:status |
MEDLINE
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pubmed:month |
Jun
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pubmed:issn |
1872-8464
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pubmed:author | |
pubmed:issnType |
Electronic
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pubmed:volume |
73
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
885-7
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pubmed:meshHeading |
pubmed-meshheading:19321208-Bronchoscopy,
pubmed-meshheading:19321208-Continuous Positive Airway Pressure,
pubmed-meshheading:19321208-Humans,
pubmed-meshheading:19321208-Hydrocephalus,
pubmed-meshheading:19321208-Intracranial Hypertension,
pubmed-meshheading:19321208-Male,
pubmed-meshheading:19321208-Tracheomalacia
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pubmed:year |
2009
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pubmed:articleTitle |
A neurologic etiology for tracheomalacia?
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pubmed:affiliation |
Department of Otorhinolaryngology-Head and Neck Surgery, Albert Einstein College of Medicine, Bronx, NY 10467, USA.
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pubmed:publicationType |
Journal Article,
Case Reports
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