| pubmed-article:19251209 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:19251209 | lifeskim:mentions | umls-concept:C0332307 | lld:lifeskim |
| pubmed-article:19251209 | lifeskim:mentions | umls-concept:C0026882 | lld:lifeskim |
| pubmed-article:19251209 | lifeskim:mentions | umls-concept:C0037473 | lld:lifeskim |
| pubmed-article:19251209 | lifeskim:mentions | umls-concept:C1720983 | lld:lifeskim |
| pubmed-article:19251209 | lifeskim:mentions | umls-concept:C0439793 | lld:lifeskim |
| pubmed-article:19251209 | lifeskim:mentions | umls-concept:C1419864 | lld:lifeskim |
| pubmed-article:19251209 | lifeskim:mentions | umls-concept:C0542560 | lld:lifeskim |
| pubmed-article:19251209 | lifeskim:mentions | umls-concept:C0205210 | lld:lifeskim |
| pubmed-article:19251209 | lifeskim:mentions | umls-concept:C0457405 | lld:lifeskim |
| pubmed-article:19251209 | pubmed:issue | 3 | lld:pubmed |
| pubmed-article:19251209 | pubmed:dateCreated | 2009-3-2 | lld:pubmed |
| pubmed-article:19251209 | pubmed:abstractText | Patients carrying loss-of-function SCN5A mutations linked to Brugada syndrome (BrS) or progressive cardiac conduction disease (PCCD) are at risk of sudden cardiac death at a young age. The penetrance and expressivity of the disease are highly variable, and new tools for risk stratification are needed. | lld:pubmed |
| pubmed-article:19251209 | pubmed:language | eng | lld:pubmed |
| pubmed-article:19251209 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:19251209 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:19251209 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:19251209 | pubmed:month | Mar | lld:pubmed |
| pubmed-article:19251209 | pubmed:issn | 1556-3871 | lld:pubmed |
| pubmed-article:19251209 | pubmed:author | pubmed-author:TanHanno LHL | lld:pubmed |
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| pubmed-article:19251209 | pubmed:author | pubmed-author:AlbuissonJJ | lld:pubmed |
| pubmed-article:19251209 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:19251209 | pubmed:volume | 6 | lld:pubmed |
| pubmed-article:19251209 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:19251209 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:19251209 | pubmed:pagination | 341-8 | lld:pubmed |
| pubmed-article:19251209 | pubmed:dateRevised | 2011-7-22 | lld:pubmed |
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| pubmed-article:19251209 | pubmed:year | 2009 | lld:pubmed |
| pubmed-article:19251209 | pubmed:articleTitle | Type of SCN5A mutation determines clinical severity and degree of conduction slowing in loss-of-function sodium channelopathies. | lld:pubmed |
| pubmed-article:19251209 | pubmed:affiliation | Department of Cardiology, Academic Medical Centre, University of Amsterdam, Amsterdam, the Netherlands. | lld:pubmed |
| pubmed-article:19251209 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:19251209 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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