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| pubmed-article:1923759 | lifeskim:mentions | umls-concept:C0008633 | lld:lifeskim |
| pubmed-article:1923759 | lifeskim:mentions | umls-concept:C0017337 | lld:lifeskim |
| pubmed-article:1923759 | lifeskim:mentions | umls-concept:C0026882 | lld:lifeskim |
| pubmed-article:1923759 | lifeskim:mentions | umls-concept:C0037067 | lld:lifeskim |
| pubmed-article:1923759 | lifeskim:mentions | umls-concept:C0205396 | lld:lifeskim |
| pubmed-article:1923759 | lifeskim:mentions | umls-concept:C2700640 | lld:lifeskim |
| pubmed-article:1923759 | lifeskim:mentions | umls-concept:C1458176 | lld:lifeskim |
| pubmed-article:1923759 | pubmed:issue | 18 | lld:pubmed |
| pubmed-article:1923759 | pubmed:dateCreated | 1991-11-4 | lld:pubmed |
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| pubmed-article:1923759 | pubmed:abstractText | The Staphylococcus aureus chromosomal gene plaC, identified by mutations such as plaC1 that lead to the amplification of plasmid pT181, has been cloned and sequenced. The plaC gene encodes a protein with high similarity (79% identity) with the vegetative sigma factor of Bacillus subtilis, sigA, suggesting that it acts as an RNA polymerase sigma factor in S.aureus. The plaC1 mutation was found to be a C to T transition leading to a proline to serine substitution at amino acid residue 209 of the protein. In other sigma factors this region of the protein is involved in specific recognition of the -10 promoter sequence. The change in sigma factor activity due to this mutation is characterized by its strict specificity for a limited number of promoters and the rather high amplitude of the effect. | lld:pubmed |
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| pubmed-article:1923759 | pubmed:language | eng | lld:pubmed |
| pubmed-article:1923759 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:1923759 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:1923759 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:1923759 | pubmed:month | Sep | lld:pubmed |
| pubmed-article:1923759 | pubmed:issn | 0305-1048 | lld:pubmed |
| pubmed-article:1923759 | pubmed:author | pubmed-author:IordanescuSS | lld:pubmed |
| pubmed-article:1923759 | pubmed:author | pubmed-author:BasheerRR | lld:pubmed |
| pubmed-article:1923759 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:1923759 | pubmed:day | 25 | lld:pubmed |
| pubmed-article:1923759 | pubmed:volume | 19 | lld:pubmed |
| pubmed-article:1923759 | pubmed:geneSymbol | plaC | lld:pubmed |
| pubmed-article:1923759 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:1923759 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:1923759 | pubmed:pagination | 4921-4 | lld:pubmed |
| pubmed-article:1923759 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:1923759 | pubmed:year | 1991 | lld:pubmed |
| pubmed-article:1923759 | pubmed:articleTitle | The Staphylococcus aureus chromosomal gene plaC, identified by mutations amplifying plasmid pT181, encodes a sigma factor. | lld:pubmed |
| pubmed-article:1923759 | pubmed:affiliation | Public Health Research Institute, New York, NY 10016. | lld:pubmed |
| pubmed-article:1923759 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:1923759 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:1923759 | pubmed:publicationType | Research Support, U.S. Gov't, Non-P.H.S. | lld:pubmed |
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