Linked Life Data

19201774

Source:http://linkedlifedata.com/resource/pubmed/id/19201774

Statements in which the resource exists.
SubjectPredicateObjectContext
pubmed-article:19201774rdf:typepubmed:Citationlld:pubmed
pubmed-article:19201774lifeskim:mentionsumls-concept:C0034693lld:lifeskim
pubmed-article:19201774lifeskim:mentionsumls-concept:C2340138lld:lifeskim
pubmed-article:19201774lifeskim:mentionsumls-concept:C0162638lld:lifeskim
pubmed-article:19201774lifeskim:mentionsumls-concept:C0239946lld:lifeskim
pubmed-article:19201774lifeskim:mentionsumls-concept:C0021467lld:lifeskim
pubmed-article:19201774lifeskim:mentionsumls-concept:C0538927lld:lifeskim
pubmed-article:19201774lifeskim:mentionsumls-concept:C0021469lld:lifeskim
pubmed-article:19201774lifeskim:mentionsumls-concept:C0205263lld:lifeskim
pubmed-article:19201774lifeskim:mentionsumls-concept:C1879547lld:lifeskim
pubmed-article:19201774pubmed:issue11lld:pubmed
pubmed-article:19201774pubmed:dateCreated2009-10-16lld:pubmed
pubmed-article:19201774pubmed:abstractTextActivated hepatic stellate cells (HSCs) but not quiescent HSCs express cyclo-oxygenase-2 (COX-2), suggesting that the COX-2/prostanoid pathway has an active role in hepatic fibrogenesis. However, the role of COX-2 inhibitors in hepatic fibrogenesis remains controversial. The aim of this study was to investigate the antifibrotic effects of celecoxib, a selective COX-2 inhibitor.lld:pubmed
pubmed-article:19201774pubmed:languageenglld:pubmed
pubmed-article:19201774pubmed:journalhttp://linkedlifedata.com/r...lld:pubmed
pubmed-article:19201774pubmed:citationSubsetAIMlld:pubmed
pubmed-article:19201774pubmed:chemicalhttp://linkedlifedata.com/r...lld:pubmed
pubmed-article:19201774pubmed:chemicalhttp://linkedlifedata.com/r...lld:pubmed
pubmed-article:19201774pubmed:chemicalhttp://linkedlifedata.com/r...lld:pubmed
pubmed-article:19201774pubmed:chemicalhttp://linkedlifedata.com/r...lld:pubmed
pubmed-article:19201774pubmed:statusMEDLINElld:pubmed
pubmed-article:19201774pubmed:monthNovlld:pubmed
pubmed-article:19201774pubmed:issn1468-3288lld:pubmed
pubmed-article:19201774pubmed:authorpubmed-author:LeeK SKSlld:pubmed
pubmed-article:19201774pubmed:authorpubmed-author:KimJ KJKlld:pubmed
pubmed-article:19201774pubmed:authorpubmed-author:LeeJ IJIlld:pubmed
pubmed-article:19201774pubmed:authorpubmed-author:LeeS ISIlld:pubmed
pubmed-article:19201774pubmed:authorpubmed-author:LeeS JSJlld:pubmed
pubmed-article:19201774pubmed:authorpubmed-author:LeeD KDKlld:pubmed
pubmed-article:19201774pubmed:authorpubmed-author:AnS HSHlld:pubmed
pubmed-article:19201774pubmed:authorpubmed-author:BrennerD ADAlld:pubmed
pubmed-article:19201774pubmed:authorpubmed-author:ORRR JRJlld:pubmed
pubmed-article:19201774pubmed:authorpubmed-author:ChoyC TCTlld:pubmed
pubmed-article:19201774pubmed:authorpubmed-author:KimD YDYlld:pubmed
pubmed-article:19201774pubmed:authorpubmed-author:KangS HSHlld:pubmed
pubmed-article:19201774pubmed:authorpubmed-author:PaikY-HYHlld:pubmed
pubmed-article:19201774pubmed:issnTypeElectroniclld:pubmed
pubmed-article:19201774pubmed:volume58lld:pubmed
pubmed-article:19201774pubmed:ownerNLMlld:pubmed
pubmed-article:19201774pubmed:authorsCompleteYlld:pubmed
pubmed-article:19201774pubmed:pagination1517-27lld:pubmed
pubmed-article:19201774pubmed:meshHeadingpubmed-meshheading:19201774...lld:pubmed
pubmed-article:19201774pubmed:meshHeadingpubmed-meshheading:19201774...lld:pubmed
pubmed-article:19201774pubmed:meshHeadingpubmed-meshheading:19201774...lld:pubmed
pubmed-article:19201774pubmed:meshHeadingpubmed-meshheading:19201774...lld:pubmed
pubmed-article:19201774pubmed:meshHeadingpubmed-meshheading:19201774...lld:pubmed
pubmed-article:19201774pubmed:meshHeadingpubmed-meshheading:19201774...lld:pubmed
pubmed-article:19201774pubmed:meshHeadingpubmed-meshheading:19201774...lld:pubmed
pubmed-article:19201774pubmed:meshHeadingpubmed-meshheading:19201774...lld:pubmed
pubmed-article:19201774pubmed:meshHeadingpubmed-meshheading:19201774...lld:pubmed
pubmed-article:19201774pubmed:meshHeadingpubmed-meshheading:19201774...lld:pubmed
pubmed-article:19201774pubmed:meshHeadingpubmed-meshheading:19201774...lld:pubmed
pubmed-article:19201774pubmed:year2009lld:pubmed
pubmed-article:19201774pubmed:articleTitleCelecoxib induces hepatic stellate cell apoptosis through inhibition of Akt activation and suppresses hepatic fibrosis in rats.lld:pubmed
pubmed-article:19201774pubmed:affiliationDepartment of Internal Medicine, Institute of Gastroenterology, Brain Korea 21 Project for Medical Science, Liver Cirrhosis Clinical Research Center, Yonsei University College of Medicine, Seoul 135-720, Korea.lld:pubmed
pubmed-article:19201774pubmed:publicationTypeJournal Articlelld:pubmed
pubmed-article:19201774pubmed:publicationTypeResearch Support, Non-U.S. Gov'tlld:pubmed
http://linkedlifedata.com/r...pubmed:referesTopubmed-article:19201774lld:pubmed
http://linkedlifedata.com/r...pubmed:referesTopubmed-article:19201774lld:pubmed
http://linkedlifedata.com/r...pubmed:referesTopubmed-article:19201774lld:pubmed