| pubmed-article:19189654 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:19189654 | lifeskim:mentions | umls-concept:C0032659 | lld:lifeskim |
| pubmed-article:19189654 | lifeskim:mentions | umls-concept:C1556094 | lld:lifeskim |
| pubmed-article:19189654 | lifeskim:mentions | umls-concept:C1366550 | lld:lifeskim |
| pubmed-article:19189654 | lifeskim:mentions | umls-concept:C1706339 | lld:lifeskim |
| pubmed-article:19189654 | lifeskim:mentions | umls-concept:C1954276 | lld:lifeskim |
| pubmed-article:19189654 | lifeskim:mentions | umls-concept:C1882417 | lld:lifeskim |
| pubmed-article:19189654 | lifeskim:mentions | umls-concept:C1314939 | lld:lifeskim |
| pubmed-article:19189654 | pubmed:issue | 6A | lld:pubmed |
| pubmed-article:19189654 | pubmed:dateCreated | 2009-2-4 | lld:pubmed |
| pubmed-article:19189654 | pubmed:abstractText | GNAS1 encodes the a-subunit of the Gs protein (Gsa), which binds GTP and stimulates adenylyl cyclase. Activating mutations lead to somatotroph, thyroid, adrenal and gonadal adenomas or the McCune-Albright syndrome and recently the T399C polymorphism in GNAS1 has been reported to be associated with malignancies. The purpose of the present case-control study with 349 Japanese prostate cancer patients and 203 urological controls was to determine whether the GNAS1 T393C polymorphism is associated with prostate cancer risk. | lld:pubmed |
| pubmed-article:19189654 | pubmed:language | eng | lld:pubmed |
| pubmed-article:19189654 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:19189654 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:19189654 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:19189654 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:19189654 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:19189654 | pubmed:issn | 0250-7005 | lld:pubmed |
| pubmed-article:19189654 | pubmed:author | pubmed-author:ShiraishiTaiz... | lld:pubmed |
| pubmed-article:19189654 | pubmed:author | pubmed-author:SuzukiHiroyos... | lld:pubmed |
| pubmed-article:19189654 | pubmed:author | pubmed-author:IchikawaTomoh... | lld:pubmed |
| pubmed-article:19189654 | pubmed:author | pubmed-author:SugimuraYoshi... | lld:pubmed |
| pubmed-article:19189654 | pubmed:author | pubmed-author:WatanabeMasat... | lld:pubmed |
| pubmed-article:19189654 | pubmed:author | pubmed-author:YanagawaMakot... | lld:pubmed |
| pubmed-article:19189654 | pubmed:author | pubmed-author:MurataTetsuya... | lld:pubmed |
| pubmed-article:19189654 | pubmed:author | pubmed-author:KatohTakahiko... | lld:pubmed |
| pubmed-article:19189654 | pubmed:author | pubmed-author:HirokawaYoshi... | lld:pubmed |
| pubmed-article:19189654 | pubmed:author | pubmed-author:TsujiMayumiM | lld:pubmed |
| pubmed-article:19189654 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:19189654 | pubmed:volume | 28 | lld:pubmed |
| pubmed-article:19189654 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:19189654 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:19189654 | pubmed:pagination | 3711-6 | lld:pubmed |
| pubmed-article:19189654 | pubmed:dateRevised | 2009-11-19 | lld:pubmed |
| pubmed-article:19189654 | pubmed:meshHeading | pubmed-meshheading:19189654... | lld:pubmed |
| pubmed-article:19189654 | pubmed:meshHeading | pubmed-meshheading:19189654... | lld:pubmed |
| pubmed-article:19189654 | pubmed:meshHeading | pubmed-meshheading:19189654... | lld:pubmed |
| pubmed-article:19189654 | pubmed:meshHeading | pubmed-meshheading:19189654... | lld:pubmed |
| pubmed-article:19189654 | pubmed:meshHeading | pubmed-meshheading:19189654... | lld:pubmed |
| pubmed-article:19189654 | pubmed:meshHeading | pubmed-meshheading:19189654... | lld:pubmed |
| pubmed-article:19189654 | pubmed:meshHeading | pubmed-meshheading:19189654... | lld:pubmed |
| pubmed-article:19189654 | pubmed:meshHeading | pubmed-meshheading:19189654... | lld:pubmed |
| pubmed-article:19189654 | pubmed:meshHeading | pubmed-meshheading:19189654... | lld:pubmed |
| pubmed-article:19189654 | pubmed:meshHeading | pubmed-meshheading:19189654... | lld:pubmed |
| pubmed-article:19189654 | pubmed:articleTitle | Lack of involvement of the GNAS1 T393C polymorphism in prostate cancer risk in a Japanese population. | lld:pubmed |
| pubmed-article:19189654 | pubmed:affiliation | Department of Pathologic Oncology, Institute of Molecular and Experimental Medicine, Mie University Graduate School of Medicine, 2-174 Edobashi, Tsu, Japan. | lld:pubmed |
| pubmed-article:19189654 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:19189654 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| entrez-gene:2778 | entrezgene:pubmed | pubmed-article:19189654 | lld:entrezgene |
| http://linkedlifedata.com/r... | entrezgene:pubmed | pubmed-article:19189654 | lld:entrezgene |
| http://linkedlifedata.com/r... | entrezgene:pubmed | pubmed-article:19189654 | lld:entrezgene |
| lhgdn:association:6232 | lhgdn:found_in | pubmed-article:19189654 | lld:lhgdn |
| lhgdn:association:6265 | lhgdn:found_in | pubmed-article:19189654 | lld:lhgdn |
