Source:http://linkedlifedata.com/resource/pubmed/id/19167040
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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
3
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pubmed:dateCreated |
2009-3-2
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pubmed:abstractText |
To examine the cellular, plasma, and urinary oxalate and erythrocyte oxalate flux in patients with calcium oxalate monohydrate (COM) stone formation vs normal controls. Pathologic oxalate clearance in humans is mostly integrated in calcium oxalate stone formation. An underlying cause of deficient oxalate clearance could be defective transmembrane oxalate transport, which, in many tissues, is regulated by an anion exchanger (SLC26).
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pubmed:language |
eng
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pubmed:journal | |
pubmed:citationSubset |
IM
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pubmed:chemical | |
pubmed:status |
MEDLINE
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pubmed:month |
Mar
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pubmed:issn |
1527-9995
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pubmed:author | |
pubmed:issnType |
Electronic
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pubmed:volume |
73
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
480-3
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pubmed:meshHeading |
pubmed-meshheading:19167040-Calcium Oxalate,
pubmed-meshheading:19167040-Cells,
pubmed-meshheading:19167040-Female,
pubmed-meshheading:19167040-Humans,
pubmed-meshheading:19167040-Male,
pubmed-meshheading:19167040-Middle Aged,
pubmed-meshheading:19167040-Oxalates,
pubmed-meshheading:19167040-Urinary Calculi
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pubmed:year |
2009
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pubmed:articleTitle |
Role of cellular oxalate in oxalate clearance of patients with calcium oxalate monohydrate stone formation and normal controls.
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pubmed:affiliation |
Department of Urology, University Hospital Carl-Gustav-Carus Technical University, Dresden, Germany. sven.oehlschläger@uniklinikum-dresden.de
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pubmed:publicationType |
Journal Article
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