rdf:type |
|
lifeskim:mentions |
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pubmed:issue |
6
|
pubmed:dateCreated |
2008-12-29
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pubmed:abstractText |
Prematurely born infants are at risk for development of neurocognitive impairment in later life. Oxygen treatment has been recently identified as a trigger of neuronal and oligodendrocyte apoptosis in the developing rodent brain. We investigated the role of the Fas death receptor pathway in oxygen-triggered developmental brain injury.
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pubmed:language |
eng
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pubmed:journal |
|
pubmed:citationSubset |
IM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Dec
|
pubmed:issn |
1531-8249
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pubmed:author |
pubmed-author:BörnerConstanzeC,
pubmed-author:BührerChristophC,
pubmed-author:BoosVinzenzV,
pubmed-author:ChauvierDavidD,
pubmed-author:DzietkoMarkM,
pubmed-author:EndesfelderStefanieS,
pubmed-author:Felderhoff-MueserUrsulaU,
pubmed-author:GenzKerstinK,
pubmed-author:GerstnerBettinaB,
pubmed-author:JacototEtienneE,
pubmed-author:ObladenMichaelM,
pubmed-author:PolleyOliverO,
pubmed-author:SifringerMarcoM
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pubmed:issnType |
Electronic
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pubmed:volume |
64
|
pubmed:owner |
NLM
|
pubmed:authorsComplete |
Y
|
pubmed:pagination |
664-73
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pubmed:meshHeading |
pubmed-meshheading:19107989-Animals,
pubmed-meshheading:19107989-Animals, Newborn,
pubmed-meshheading:19107989-Brain Injuries,
pubmed-meshheading:19107989-Fas Ligand Protein,
pubmed-meshheading:19107989-Hyperoxia,
pubmed-meshheading:19107989-Mice,
pubmed-meshheading:19107989-Mice, Inbred C57BL,
pubmed-meshheading:19107989-Mice, Transgenic,
pubmed-meshheading:19107989-Rats,
pubmed-meshheading:19107989-Rats, Wistar,
pubmed-meshheading:19107989-Signal Transduction
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pubmed:year |
2008
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pubmed:articleTitle |
A critical role for Fas/CD-95 dependent signaling pathways in the pathogenesis of hyperoxia-induced brain injury.
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pubmed:affiliation |
Department of Neonatology, Charité, Universitätsmedizin-Berlin, Campus Virchow Klinikum, Berlin, Germany. mark.dzietko@charite.de
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pubmed:publicationType |
Journal Article,
Comparative Study,
Research Support, Non-U.S. Gov't
|