Source:http://linkedlifedata.com/resource/pubmed/id/19102398
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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
86-87
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pubmed:dateCreated |
2008-12-23
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pubmed:abstractText |
Irrespective of the underlying etiology the immune response to is almost identical in severe cases of acute pancreatitis. While the triggering factors of acute pancreatitis are still poorly understood, cytokines are considered as important mediators in the pathophysiology of severe acute pancreatitis. However, only few studies have investigated the role of IL-18 and ICAM-1 in human acute pancreatitis.
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pubmed:language |
eng
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pubmed:journal | |
pubmed:citationSubset |
IM
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pubmed:chemical | |
pubmed:status |
MEDLINE
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pubmed:issn |
0172-6390
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pubmed:author | |
pubmed:issnType |
Print
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pubmed:volume |
55
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
1806-13
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pubmed:meshHeading |
pubmed-meshheading:19102398-Acute Disease,
pubmed-meshheading:19102398-Adult,
pubmed-meshheading:19102398-Female,
pubmed-meshheading:19102398-Humans,
pubmed-meshheading:19102398-Intercellular Adhesion Molecule-1,
pubmed-meshheading:19102398-Interleukin-18,
pubmed-meshheading:19102398-Male,
pubmed-meshheading:19102398-Middle Aged,
pubmed-meshheading:19102398-Pancreatitis
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pubmed:articleTitle |
Implication of interleukin 18 and intercellular adhesion molecule (ICAM)-1 in acute pancreatitis.
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pubmed:affiliation |
Department of Surgery, Medical University, Lviv, Ukraine. perejaslov_andre@yahoo.com
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pubmed:publicationType |
Journal Article
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