| pubmed-article:19036974 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:19036974 | lifeskim:mentions | umls-concept:C0927232 | lld:lifeskim |
| pubmed-article:19036974 | lifeskim:mentions | umls-concept:C0596991 | lld:lifeskim |
| pubmed-article:19036974 | lifeskim:mentions | umls-concept:C0007578 | lld:lifeskim |
| pubmed-article:19036974 | lifeskim:mentions | umls-concept:C1425010 | lld:lifeskim |
| pubmed-article:19036974 | lifeskim:mentions | umls-concept:C0181586 | lld:lifeskim |
| pubmed-article:19036974 | lifeskim:mentions | umls-concept:C0205421 | lld:lifeskim |
| pubmed-article:19036974 | lifeskim:mentions | umls-concept:C0332453 | lld:lifeskim |
| pubmed-article:19036974 | pubmed:issue | 48 | lld:pubmed |
| pubmed-article:19036974 | pubmed:dateCreated | 2008-11-27 | lld:pubmed |
| pubmed-article:19036974 | pubmed:abstractText | Nectin-like 1 (Necl-1) is a neural-specific cell adhesion molecule that is expressed in both the CNS and PNS. Previous in vitro studies suggested that Necl-1 expression is essential for the axon-glial interaction and myelin sheath formation in the PNS. To investigate the in vivo role of Necl-1 in axonal myelination of the developing nervous system, we generated the Necl-1 mutant mice by replacing axons 2-5 with the LacZ reporter gene. Expression studies revealed that Necl-1 is exclusively expressed by neurons in the CNS. Disruption of Necl-1 resulted in developmental delay of axonal myelination in the optic nerve and spinal cord, suggesting that Necl-1 plays an important role in the initial axon-oligodendrocyte recognition and adhesion in CNS myelination. | lld:pubmed |
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| pubmed-article:19036974 | pubmed:language | eng | lld:pubmed |
| pubmed-article:19036974 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:19036974 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:19036974 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:19036974 | pubmed:month | Nov | lld:pubmed |
| pubmed-article:19036974 | pubmed:issn | 1529-2401 | lld:pubmed |
| pubmed-article:19036974 | pubmed:author | pubmed-author:LiHongH | lld:pubmed |
| pubmed-article:19036974 | pubmed:author | pubmed-author:ZhuYingY | lld:pubmed |
| pubmed-article:19036974 | pubmed:author | pubmed-author:ZhuQiangQ | lld:pubmed |
| pubmed-article:19036974 | pubmed:author | pubmed-author:QiuMengshengM | lld:pubmed |
| pubmed-article:19036974 | pubmed:author | pubmed-author:YuanJiangangJ | lld:pubmed |
| pubmed-article:19036974 | pubmed:author | pubmed-author:QiangBoqinB | lld:pubmed |
| pubmed-article:19036974 | pubmed:author | pubmed-author:ChenTaoT | lld:pubmed |
| pubmed-article:19036974 | pubmed:author | pubmed-author:GaoJingJ | lld:pubmed |
| pubmed-article:19036974 | pubmed:author | pubmed-author:PengXiaozhong... | lld:pubmed |
| pubmed-article:19036974 | pubmed:author | pubmed-author:HuXuemeiX | lld:pubmed |
| pubmed-article:19036974 | pubmed:author | pubmed-author:FEIL MLM | lld:pubmed |
| pubmed-article:19036974 | pubmed:author | pubmed-author:ParkJinsilJ | lld:pubmed |
| pubmed-article:19036974 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:19036974 | pubmed:day | 26 | lld:pubmed |
| pubmed-article:19036974 | pubmed:volume | 28 | lld:pubmed |
| pubmed-article:19036974 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:19036974 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:19036974 | pubmed:pagination | 12815-9 | lld:pubmed |
| pubmed-article:19036974 | pubmed:dateRevised | 2011-11-17 | lld:pubmed |
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| pubmed-article:19036974 | pubmed:year | 2008 | lld:pubmed |
| pubmed-article:19036974 | pubmed:articleTitle | Disruption of Nectin-like 1 cell adhesion molecule leads to delayed axonal myelination in the CNS. | lld:pubmed |
| pubmed-article:19036974 | pubmed:affiliation | Department of Anatomical Sciences and Neurobiology, School of Medicine, University of Louisville, Louisville, Kentucky 40292, USA. | lld:pubmed |
| pubmed-article:19036974 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:19036974 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| pubmed-article:19036974 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
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