| pubmed-article:19025532 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:19025532 | lifeskim:mentions | umls-concept:C1135183 | lld:lifeskim |
| pubmed-article:19025532 | lifeskim:mentions | umls-concept:C0026336 | lld:lifeskim |
| pubmed-article:19025532 | lifeskim:mentions | umls-concept:C0022646 | lld:lifeskim |
| pubmed-article:19025532 | lifeskim:mentions | umls-concept:C0025962 | lld:lifeskim |
| pubmed-article:19025532 | lifeskim:mentions | umls-concept:C0036974 | lld:lifeskim |
| pubmed-article:19025532 | lifeskim:mentions | umls-concept:C0332161 | lld:lifeskim |
| pubmed-article:19025532 | lifeskim:mentions | umls-concept:C1134681 | lld:lifeskim |
| pubmed-article:19025532 | lifeskim:mentions | umls-concept:C0907956 | lld:lifeskim |
| pubmed-article:19025532 | pubmed:issue | 10 | lld:pubmed |
| pubmed-article:19025532 | pubmed:dateCreated | 2008-11-25 | lld:pubmed |
| pubmed-article:19025532 | pubmed:abstractText | Impaired renal microcirculation has been suggested as a factor contributing to the development of renal dysfunction in sepsis. This study was conducted to elucidate the role of endothelin-1 (ET-1)in mediating reductions in renal microcirculatory blood flow during endotoxemic shock. | lld:pubmed |
| pubmed-article:19025532 | pubmed:language | eng | lld:pubmed |
| pubmed-article:19025532 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:19025532 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:19025532 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:19025532 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:19025532 | pubmed:month | Nov | lld:pubmed |
| pubmed-article:19025532 | pubmed:issn | 1399-6576 | lld:pubmed |
| pubmed-article:19025532 | pubmed:author | pubmed-author:AnderssonAA | lld:pubmed |
| pubmed-article:19025532 | pubmed:author | pubmed-author:ForestierJJ | lld:pubmed |
| pubmed-article:19025532 | pubmed:author | pubmed-author:SolleviAA | lld:pubmed |
| pubmed-article:19025532 | pubmed:author | pubmed-author:WeitzbergEE | lld:pubmed |
| pubmed-article:19025532 | pubmed:author | pubmed-author:HjelmqvistHH | lld:pubmed |
| pubmed-article:19025532 | pubmed:author | pubmed-author:FrithiofRR | lld:pubmed |
| pubmed-article:19025532 | pubmed:author | pubmed-author:FenhammarJJ | lld:pubmed |
| pubmed-article:19025532 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:19025532 | pubmed:volume | 52 | lld:pubmed |
| pubmed-article:19025532 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:19025532 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:19025532 | pubmed:pagination | 1385-93 | lld:pubmed |
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| pubmed-article:19025532 | pubmed:year | 2008 | lld:pubmed |
| pubmed-article:19025532 | pubmed:articleTitle | The endothelin receptor antagonist tezosentan improves renal microcirculation in a porcine model of endotoxemic shock. | lld:pubmed |
| pubmed-article:19025532 | pubmed:affiliation | Department of Anaesthesia and Intensive Care, Karolinska University Hospital, Stockholm, Sweden. johan.fenhammar@karolinska.se | lld:pubmed |
| pubmed-article:19025532 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:19025532 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:19025532 | lld:pubmed |
