19023100

Source:http://linkedlifedata.com/resource/pubmed/id/19023100

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Predicate	Object
rdf:type	pubmed:Citation
lifeskim:mentions	umls-concept:C0003017, umls-concept:C0017337, umls-concept:C0025936, umls-concept:C0035094, umls-concept:C0086418, umls-concept:C0205234, umls-concept:C0206243, umls-concept:C0917798
pubmed:issue	2
pubmed:dateCreated	2009-1-27
pubmed:abstractText	We examined the possibility that activation of the human brain renin-angiotensin system is involved in enhancement of ischemic brain damage using chimeric transgenic mice with human renin (hRN) and human angiotensinogen (hANG) genes.
pubmed:language	eng
pubmed:journal	http://linkedlifedata.com/resource/pubmed/journal/0235266
pubmed:citationSubset	IM
pubmed:chemical	http://linkedlifedata.com/resource/pubmed/chemical/Angiotensin II, http://linkedlifedata.com/resource/pubmed/chemical/Angiotensin II Type 1 Receptor..., http://linkedlifedata.com/resource/pubmed/chemical/Angiotensinogen, http://linkedlifedata.com/resource/pubmed/chemical/Receptor, Angiotensin, Type 1, http://linkedlifedata.com/resource/pubmed/chemical/Receptor, Angiotensin, Type 2, http://linkedlifedata.com/resource/pubmed/chemical/Renin, http://linkedlifedata.com/resource/pubmed/chemical/Superoxides, http://linkedlifedata.com/resource/pubmed/chemical/Tetrazoles, http://linkedlifedata.com/resource/pubmed/chemical/Valine, http://linkedlifedata.com/resource/pubmed/chemical/valsartan
pubmed:status	MEDLINE
pubmed:month	Feb
pubmed:issn	1524-4628
pubmed:author	pubmed-author:FurunoMegumiM, pubmed-author:HigakiJitsuoJ, pubmed-author:HoriuchiMasatsuguM, pubmed-author:InabaShinjiS, pubmed-author:IwaiMasaruM, pubmed-author:KannoHarumiH, pubmed-author:MogiMasakiM, pubmed-author:OkayamaHidekiH, pubmed-author:SenbaIzumiI, pubmed-author:TomonoYumikoY
pubmed:issnType	Electronic
pubmed:volume	40
pubmed:owner	NLM
pubmed:authorsComplete	Y
pubmed:pagination	597-603
pubmed:meshHeading	pubmed-meshheading:19023100-Angiotensin II, pubmed-meshheading:19023100-Angiotensin II Type 1 Receptor Blockers, pubmed-meshheading:19023100-Angiotensinogen, pubmed-meshheading:19023100-Animals, pubmed-meshheading:19023100-Brain Chemistry, pubmed-meshheading:19023100-Brain Ischemia, pubmed-meshheading:19023100-Capillaries, pubmed-meshheading:19023100-Cerebrovascular Circulation, pubmed-meshheading:19023100-Humans, pubmed-meshheading:19023100-Infarction, Middle Cerebral Artery, pubmed-meshheading:19023100-Male, pubmed-meshheading:19023100-Mice, pubmed-meshheading:19023100-Mice, Transgenic, pubmed-meshheading:19023100-Oxidative Stress, pubmed-meshheading:19023100-Receptor, Angiotensin, Type 1, pubmed-meshheading:19023100-Receptor, Angiotensin, Type 2, pubmed-meshheading:19023100-Renin, pubmed-meshheading:19023100-Reverse Transcriptase Polymerase Chain Reaction, pubmed-meshheading:19023100-Superoxides, pubmed-meshheading:19023100-Tetrazoles, pubmed-meshheading:19023100-Valine
pubmed:year	2009
pubmed:articleTitle	Exaggeration of focal cerebral ischemia in transgenic mice carrying human Renin and human angiotensinogen genes.
pubmed:affiliation	Department of Molecular Cardiovascular Biology and Pharmacology, Ehime University Graduate School of Medicine, Shitsukawa, Tohon, Ehime, Japan.
pubmed:publicationType	Journal Article, Research Support, Non-U.S. Gov't