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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
14
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pubmed:dateCreated |
2009-4-7
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pubmed:abstractText |
Dyskinesias are common in Parkinson disease (PD). Prior investigations suggest that dopamine (DA) terminals compensate for abnormal DA transmission. We verified whether similar adaptations could be related to the development of treatment-related complications.
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pubmed:commentsCorrections |
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
AIM
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pubmed:chemical |
|
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pubmed:status |
MEDLINE
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pubmed:month |
Apr
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pubmed:issn |
1526-632X
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pubmed:author |
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pubmed:issnType |
Electronic
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pubmed:day |
7
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pubmed:volume |
72
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
1211-6
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pubmed:meshHeading |
pubmed-meshheading:19020294-Caudate Nucleus,
pubmed-meshheading:19020294-Dopamine Plasma Membrane Transport Proteins,
pubmed-meshheading:19020294-Dyskinesias,
pubmed-meshheading:19020294-Female,
pubmed-meshheading:19020294-Humans,
pubmed-meshheading:19020294-Logistic Models,
pubmed-meshheading:19020294-Male,
pubmed-meshheading:19020294-Methylphenidate,
pubmed-meshheading:19020294-Middle Aged,
pubmed-meshheading:19020294-Parkinson Disease,
pubmed-meshheading:19020294-Positron-Emission Tomography,
pubmed-meshheading:19020294-Putamen,
pubmed-meshheading:19020294-Radiopharmaceuticals,
pubmed-meshheading:19020294-Tetrabenazine
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pubmed:year |
2009
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pubmed:articleTitle |
PET demonstrates reduced dopamine transporter expression in PD with dyskinesias.
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pubmed:affiliation |
Pacific Parkinson's Research Centre, Vancouver, BC, Canada. andretroiano@gmail.com
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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