|
rdf:type |
|
|
lifeskim:mentions |
umls-concept:C0003507,
umls-concept:C0014257,
umls-concept:C0030705,
umls-concept:C0038250,
umls-concept:C0043240,
umls-concept:C0178566,
umls-concept:C0205314,
umls-concept:C0225336,
umls-concept:C0237753,
umls-concept:C0374711,
umls-concept:C0392756,
umls-concept:C0542341,
umls-concept:C0679622,
umls-concept:C1705181
|
|
pubmed:issue |
3
|
|
pubmed:dateCreated |
2009-2-2
|
|
pubmed:abstractText |
Endothelial destruction and calcification primarily occur at the aortic side of the calcified aortic valves (AVs). This study investigated whether degenerative AV stenosis (AS) is associated with the presence of valvular endothelial senescence and a reduction in the number and function of endothelial progenitor cells (EPCs).
|
|
pubmed:language |
eng
|
|
pubmed:journal |
|
|
pubmed:citationSubset |
IM
|
|
pubmed:chemical |
|
|
pubmed:status |
MEDLINE
|
|
pubmed:month |
Feb
|
|
pubmed:issn |
1522-9645
|
|
pubmed:author |
|
|
pubmed:issnType |
Electronic
|
|
pubmed:volume |
30
|
|
pubmed:owner |
NLM
|
|
pubmed:authorsComplete |
Y
|
|
pubmed:pagination |
346-55
|
|
pubmed:meshHeading |
pubmed-meshheading:19010796-Aged,
pubmed-meshheading:19010796-Antigens, CD34,
pubmed-meshheading:19010796-Aortic Valve Stenosis,
pubmed-meshheading:19010796-Apoptosis,
pubmed-meshheading:19010796-Calcinosis,
pubmed-meshheading:19010796-Caspase 3,
pubmed-meshheading:19010796-Cell Aging,
pubmed-meshheading:19010796-Cell Movement,
pubmed-meshheading:19010796-Cells, Cultured,
pubmed-meshheading:19010796-Disease Progression,
pubmed-meshheading:19010796-Echocardiography, Doppler,
pubmed-meshheading:19010796-Endothelium, Vascular,
pubmed-meshheading:19010796-Female,
pubmed-meshheading:19010796-Heart Catheterization,
pubmed-meshheading:19010796-Humans,
pubmed-meshheading:19010796-Kruppel-Like Transcription Factors,
pubmed-meshheading:19010796-Male,
pubmed-meshheading:19010796-Middle Aged,
pubmed-meshheading:19010796-Stem Cells,
pubmed-meshheading:19010796-Telomeric Repeat Binding Protein 2
|
|
pubmed:year |
2009
|
|
pubmed:articleTitle |
Reduced number and function of endothelial progenitor cells in patients with aortic valve stenosis: a novel concept for valvular endothelial cell repair.
|
|
pubmed:affiliation |
Department of Cardiology, Heart Center Leipzig, University of Leipzig, Strümpellstrasse 39, D-04289 Leipzig, Germany.
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|
pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
|
