Source:http://linkedlifedata.com/resource/pubmed/id/19008173
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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
10
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pubmed:dateCreated |
2008-11-14
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pubmed:abstractText |
The intrathecal posttraumatic inflammation contributes to secondary brain damage as well as to the induction of neuroreparative mechanisms. In this context release of interleukin-10 (IL-10) has been reported to play a major role. However, initial IL-10 concentration in CSF remains incompletely characterized. Therefore, the aim was to analyze Il-10 in CSF and serum of patients early after TBI.
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pubmed:language |
eng
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pubmed:journal | |
pubmed:citationSubset |
IM
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pubmed:chemical | |
pubmed:status |
MEDLINE
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pubmed:month |
Oct
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pubmed:issn |
0949-2321
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pubmed:author | |
pubmed:issnType |
Print
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pubmed:day |
27
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pubmed:volume |
13
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
464-8
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pubmed:meshHeading |
pubmed-meshheading:19008173-Adult,
pubmed-meshheading:19008173-Blood-Brain Barrier,
pubmed-meshheading:19008173-Brain Injuries,
pubmed-meshheading:19008173-Case-Control Studies,
pubmed-meshheading:19008173-Female,
pubmed-meshheading:19008173-Humans,
pubmed-meshheading:19008173-Inflammation Mediators,
pubmed-meshheading:19008173-Interleukin-10,
pubmed-meshheading:19008173-Male,
pubmed-meshheading:19008173-Middle Aged,
pubmed-meshheading:19008173-Prognosis
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pubmed:year |
2008
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pubmed:articleTitle |
Cerebrospinal IL-10 concentration is elevated in non-survivors as compared to survivors after severe traumatic brain injury.
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pubmed:affiliation |
Abteilung für Sportorthopädie, Klinikum Rechts der Isar, Technische Universität München, Conollystrasse 32, 80809 München, Germany. Chlodwig.Kirchhoff@med.uni-muenchen.de
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pubmed:publicationType |
Journal Article,
Comparative Study
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