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| pubmed-article:18982072 | lifeskim:mentions | umls-concept:C0442108 | lld:lifeskim |
| pubmed-article:18982072 | lifeskim:mentions | umls-concept:C0287186 | lld:lifeskim |
| pubmed-article:18982072 | lifeskim:mentions | umls-concept:C0004379 | lld:lifeskim |
| pubmed-article:18982072 | lifeskim:mentions | umls-concept:C0449258 | lld:lifeskim |
| pubmed-article:18982072 | lifeskim:mentions | umls-concept:C0017262 | lld:lifeskim |
| pubmed-article:18982072 | lifeskim:mentions | umls-concept:C0023688 | lld:lifeskim |
| pubmed-article:18982072 | lifeskim:mentions | umls-concept:C0441722 | lld:lifeskim |
| pubmed-article:18982072 | lifeskim:mentions | umls-concept:C0205263 | lld:lifeskim |
| pubmed-article:18982072 | lifeskim:mentions | umls-concept:C2911684 | lld:lifeskim |
| pubmed-article:18982072 | lifeskim:mentions | umls-concept:C0185117 | lld:lifeskim |
| pubmed-article:18982072 | pubmed:issue | 11 | lld:pubmed |
| pubmed-article:18982072 | pubmed:dateCreated | 2008-11-4 | lld:pubmed |
| pubmed-article:18982072 | pubmed:abstractText | One of the hallmarks of rheumatoid arthritis (RA) is hyperplasia and inflammation of the synovial tissue being characterized by in situ occurrence of highly differentiated leukocytes. Fms-like tyrosine kinase 3 (Flt3) has a crucial role in hematopoiesis, regulation of cell proliferation, differentiation and apoptosis. Typically, Flt3 is expressed on early myeloid and lymphoid progenitors and is activated by its soluble ligand (Flt3-L). The highly differentiated cellular pattern in the synovium of the RA patients made us hypothesize that Flt3-L, with its ability to induce proliferation and differentiation, could be of importance in induction and/or progression of arthritis. | lld:pubmed |
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| pubmed-article:18982072 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:18982072 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:18982072 | pubmed:issn | 1932-6203 | lld:pubmed |
| pubmed-article:18982072 | pubmed:author | pubmed-author:MagnussonMatt... | lld:pubmed |
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| pubmed-article:18982072 | pubmed:author | pubmed-author:DehlinMatsM | lld:pubmed |
| pubmed-article:18982072 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:18982072 | pubmed:volume | 3 | lld:pubmed |
| pubmed-article:18982072 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:18982072 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:18982072 | pubmed:pagination | e3633 | lld:pubmed |
| pubmed-article:18982072 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:18982072 | pubmed:year | 2008 | lld:pubmed |
| pubmed-article:18982072 | pubmed:articleTitle | Intra-articular fms-like tyrosine kinase 3 ligand expression is a driving force in induction and progression of arthritis. | lld:pubmed |
| pubmed-article:18982072 | pubmed:affiliation | Department of Rheumatology and Inflammation Research, Göteborg University, Göteborg, Sweden. mats.dehlin@vgregion.se | lld:pubmed |
| pubmed-article:18982072 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:18982072 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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