rdf:type |
|
lifeskim:mentions |
|
pubmed:issue |
12
|
pubmed:dateCreated |
2008-11-26
|
pubmed:abstractText |
Although the level of erythrocyte complement receptor type 1 (E-CR1) expression in patients with malaria has been extensively studied, whether the level of expression of E-CR1 is associated with severe malaria remains controversial. The present study examined a possible association of polymorphisms in the CR1 gene with the severity of malaria, and it evaluated the influence of the associated polymorphism on expression of E-CR1.
|
pubmed:language |
eng
|
pubmed:journal |
|
pubmed:citationSubset |
AIM
|
pubmed:chemical |
|
pubmed:status |
MEDLINE
|
pubmed:month |
Dec
|
pubmed:issn |
0022-1899
|
pubmed:author |
|
pubmed:issnType |
Print
|
pubmed:day |
15
|
pubmed:volume |
198
|
pubmed:owner |
NLM
|
pubmed:authorsComplete |
Y
|
pubmed:pagination |
1880-91
|
pubmed:dateRevised |
2010-11-18
|
pubmed:meshHeading |
pubmed-meshheading:18954261-Genetic Linkage,
pubmed-meshheading:18954261-Genetic Predisposition to Disease,
pubmed-meshheading:18954261-Humans,
pubmed-meshheading:18954261-Malaria, Cerebral,
pubmed-meshheading:18954261-Polymorphism, Single Nucleotide,
pubmed-meshheading:18954261-Promoter Regions, Genetic,
pubmed-meshheading:18954261-Receptors, Complement 3b
|
pubmed:year |
2008
|
pubmed:articleTitle |
A functional single-nucleotide polymorphism in the CR1 promoter region contributes to protection against cerebral malaria.
|
pubmed:affiliation |
Department of Human Genetics, Graduate School of Medicine, University of Tokyo, Tokyo, Japan.
|
pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
|