| pubmed-article:18929331 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:18929331 | lifeskim:mentions | umls-concept:C1273518 | lld:lifeskim |
| pubmed-article:18929331 | lifeskim:mentions | umls-concept:C0241888 | lld:lifeskim |
| pubmed-article:18929331 | lifeskim:mentions | umls-concept:C0004238 | lld:lifeskim |
| pubmed-article:18929331 | lifeskim:mentions | umls-concept:C0023976 | lld:lifeskim |
| pubmed-article:18929331 | lifeskim:mentions | umls-concept:C0026882 | lld:lifeskim |
| pubmed-article:18929331 | lifeskim:mentions | umls-concept:C0037492 | lld:lifeskim |
| pubmed-article:18929331 | lifeskim:mentions | umls-concept:C0004083 | lld:lifeskim |
| pubmed-article:18929331 | pubmed:issue | 10 | lld:pubmed |
| pubmed-article:18929331 | pubmed:dateCreated | 2008-10-20 | lld:pubmed |
| pubmed-article:18929331 | pubmed:abstractText | Type 3 long-QT syndrome (LQT-3) is caused by gain-of-function mutations in the SCN5A encoding the cardiac sodium channel. Familial atrial fibrillation (AF), previously considered a potassium channelopathy, has recently been related to sodium genetic variants, both in isolated forms and in patients with underlying heart disease. | lld:pubmed |
| pubmed-article:18929331 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:18929331 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:18929331 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:18929331 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:18929331 | pubmed:month | Oct | lld:pubmed |
| pubmed-article:18929331 | pubmed:issn | 1556-3871 | lld:pubmed |
| pubmed-article:18929331 | pubmed:author | pubmed-author:BrugadaJosepJ | lld:pubmed |
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| pubmed-article:18929331 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:18929331 | pubmed:volume | 5 | lld:pubmed |
| pubmed-article:18929331 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:18929331 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:18929331 | pubmed:pagination | 1434-40 | lld:pubmed |
| pubmed-article:18929331 | pubmed:dateRevised | 2011-7-22 | lld:pubmed |
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| pubmed-article:18929331 | pubmed:year | 2008 | lld:pubmed |
| pubmed-article:18929331 | pubmed:articleTitle | A mutation in the sodium channel is responsible for the association of long QT syndrome and familial atrial fibrillation. | lld:pubmed |
| pubmed-article:18929331 | pubmed:affiliation | Cardiovascular Genetics Center, Montreal Heart Institute, University of Montreal, Montreal, Canada. | lld:pubmed |
| pubmed-article:18929331 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:18929331 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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