pubmed-article:18813210 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:18813210 | lifeskim:mentions | umls-concept:C2347026 | lld:lifeskim |
pubmed-article:18813210 | lifeskim:mentions | umls-concept:C0370003 | lld:lifeskim |
pubmed-article:18813210 | lifeskim:mentions | umls-concept:C0036341 | lld:lifeskim |
pubmed-article:18813210 | lifeskim:mentions | umls-concept:C0209706 | lld:lifeskim |
pubmed-article:18813210 | lifeskim:mentions | umls-concept:C0085862 | lld:lifeskim |
pubmed-article:18813210 | lifeskim:mentions | umls-concept:C1299583 | lld:lifeskim |
pubmed-article:18813210 | lifeskim:mentions | umls-concept:C0752046 | lld:lifeskim |
pubmed-article:18813210 | lifeskim:mentions | umls-concept:C0004083 | lld:lifeskim |
pubmed-article:18813210 | lifeskim:mentions | umls-concept:C0936012 | lld:lifeskim |
pubmed-article:18813210 | lifeskim:mentions | umls-concept:C1608386 | lld:lifeskim |
pubmed-article:18813210 | lifeskim:mentions | umls-concept:C1698593 | lld:lifeskim |
pubmed-article:18813210 | lifeskim:mentions | umls-concept:C1549571 | lld:lifeskim |
pubmed-article:18813210 | pubmed:issue | 1 | lld:pubmed |
pubmed-article:18813210 | pubmed:dateCreated | 2008-12-19 | lld:pubmed |
pubmed-article:18813210 | pubmed:abstractText | We and others have previously reported linkage to schizophrenia on chromosome 10q25-q26 but, to date, a susceptibility gene in the region has not been identified. We examined data from 3606 single-nucleotide polymorphisms (SNPs) mapping to 10q25-q26 that had been typed in a genome-wide association study (GWAS) of schizophrenia (479 UK cases/2937 controls). SNPs with P<0.01 (n=40) were genotyped in an additional 163 UK cases and those markers that remained nominally significant at P<0.01 (n=22) were genotyped in replication samples from Ireland, Germany and Bulgaria consisting of a total of 1664 cases with schizophrenia and 3541 controls. Only one SNP, rs17101921, was nominally significant after meta-analyses across the replication samples and this was genotyped in an additional six samples from the United States/Australia, Germany, China, Japan, Israel and Sweden (n=5142 cases/6561 controls). Across all replication samples, the allele at rs17101921 that was associated in the GWAS showed evidence for association independent of the original data (OR 1.17 (95% CI 1.06-1.29), P=0.0009). The SNP maps 85 kb from the nearest gene encoding fibroblast growth factor receptor 2 (FGFR2) making this a potential susceptibility gene for schizophrenia. | lld:pubmed |
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pubmed-article:18813210 | pubmed:language | eng | lld:pubmed |
pubmed-article:18813210 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:18813210 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:18813210 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:18813210 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:18813210 | pubmed:month | Jan | lld:pubmed |
pubmed-article:18813210 | pubmed:issn | 1476-5578 | lld:pubmed |
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