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| pubmed-article:18796522 | lifeskim:mentions | umls-concept:C0205419 | lld:lifeskim |
| pubmed-article:18796522 | lifeskim:mentions | umls-concept:C1866503 | lld:lifeskim |
| pubmed-article:18796522 | lifeskim:mentions | umls-concept:C1416578 | lld:lifeskim |
| pubmed-article:18796522 | lifeskim:mentions | umls-concept:C0004083 | lld:lifeskim |
| pubmed-article:18796522 | lifeskim:mentions | umls-concept:C0392756 | lld:lifeskim |
| pubmed-article:18796522 | pubmed:issue | 12 | lld:pubmed |
| pubmed-article:18796522 | pubmed:dateCreated | 2008-12-5 | lld:pubmed |
| pubmed-article:18796522 | pubmed:abstractText | ATP-sensitive potassium channels are composed of pore-forming (Kir6.x) and regulatory sulfonylurea receptor (SURx) subunits and have been implicated in the maintenance of glucose homeostasis. Kir6.2 and SUR1 are expressed in a broad range of tissues, and no contemporary studies have addressed the physiological impact of variants in Hispanic-Americans and African-Americans carefully phenotyped for components of glucose homeostasis. | lld:pubmed |
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| pubmed-article:18796522 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:18796522 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:18796522 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:18796522 | pubmed:month | Dec | lld:pubmed |
| pubmed-article:18796522 | pubmed:issn | 0021-972X | lld:pubmed |
| pubmed-article:18796522 | pubmed:author | pubmed-author:Bryer-AshMich... | lld:pubmed |
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| pubmed-article:18796522 | pubmed:author | pubmed-author:LangefeldCarl... | lld:pubmed |
| pubmed-article:18796522 | pubmed:author | pubmed-author:TaylorKent... | lld:pubmed |
| pubmed-article:18796522 | pubmed:author | pubmed-author:PalmerNichole... | lld:pubmed |
| pubmed-article:18796522 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:18796522 | pubmed:volume | 93 | lld:pubmed |
| pubmed-article:18796522 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:18796522 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:18796522 | pubmed:pagination | 4979-83 | lld:pubmed |
| pubmed-article:18796522 | pubmed:dateRevised | 2011-11-17 | lld:pubmed |
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| pubmed-article:18796522 | pubmed:year | 2008 | lld:pubmed |
| pubmed-article:18796522 | pubmed:articleTitle | Association of the Kir6.2 E23K variant with reduced acute insulin response in African-Americans. | lld:pubmed |
| pubmed-article:18796522 | pubmed:affiliation | Department of Biochemistry, Wake Forest University School of Medicine, Winston-Salem, North Carolina 27157, USA. | lld:pubmed |
| pubmed-article:18796522 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:18796522 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
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