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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
12
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pubmed:dateCreated |
2008-12-5
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pubmed:abstractText |
ATP-sensitive potassium channels are composed of pore-forming (Kir6.x) and regulatory sulfonylurea receptor (SURx) subunits and have been implicated in the maintenance of glucose homeostasis. Kir6.2 and SUR1 are expressed in a broad range of tissues, and no contemporary studies have addressed the physiological impact of variants in Hispanic-Americans and African-Americans carefully phenotyped for components of glucose homeostasis.
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pubmed:grant |
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pubmed:commentsCorrections |
http://linkedlifedata.com/resource/pubmed/commentcorrection/18796522-10204114,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18796522-11815482,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18796522-11872696,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18796522-12028373,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18796522-12351459,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18796522-12540638,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18796522-12684185,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18796522-12819904,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18796522-12882937,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18796522-14639702,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18796522-15013476,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18796522-15111507,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18796522-15115830,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18796522-15842514,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18796522-15910876,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18796522-16885549,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18796522-2452599,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18796522-3640682,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18796522-3884329,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18796522-443421,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18796522-7502040,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18796522-8425661,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18796522-8596634,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18796522-9032110,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18796522-9342346,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18796522-9545414,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18796522-9634505
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
AIM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Dec
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pubmed:issn |
0021-972X
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:volume |
93
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
4979-83
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pubmed:dateRevised |
2011-11-17
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pubmed:meshHeading |
pubmed-meshheading:18796522-ATP-Binding Cassette Transporters,
pubmed-meshheading:18796522-Adult,
pubmed-meshheading:18796522-African Americans,
pubmed-meshheading:18796522-Body Mass Index,
pubmed-meshheading:18796522-Female,
pubmed-meshheading:18796522-Gene Frequency,
pubmed-meshheading:18796522-Genotype,
pubmed-meshheading:18796522-Glucose Tolerance Test,
pubmed-meshheading:18796522-Hispanic Americans,
pubmed-meshheading:18796522-Homeostasis,
pubmed-meshheading:18796522-Humans,
pubmed-meshheading:18796522-Insulin,
pubmed-meshheading:18796522-Insulin Resistance,
pubmed-meshheading:18796522-Male,
pubmed-meshheading:18796522-Mutation,
pubmed-meshheading:18796522-Potassium Channels, Inwardly Rectifying,
pubmed-meshheading:18796522-Receptors, Drug
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pubmed:year |
2008
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pubmed:articleTitle |
Association of the Kir6.2 E23K variant with reduced acute insulin response in African-Americans.
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pubmed:affiliation |
Department of Biochemistry, Wake Forest University School of Medicine, Winston-Salem, North Carolina 27157, USA.
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pubmed:publicationType |
Journal Article,
Research Support, N.I.H., Extramural
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