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rdf:type |
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lifeskim:mentions |
umls-concept:C0021655,
umls-concept:C0023796,
umls-concept:C0037083,
umls-concept:C0037663,
umls-concept:C0086418,
umls-concept:C0205178,
umls-concept:C0205263,
umls-concept:C0332291,
umls-concept:C0574032,
umls-concept:C0911014,
umls-concept:C1710082
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pubmed:issue |
12
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pubmed:dateCreated |
2008-11-26
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pubmed:databankReference |
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pubmed:abstractText |
Ghrelin is a gut-derived peptide and an endogenous ligand for the growth hormone (GH) secretagogue receptor. Exogenous ghrelin stimulates the release of GH (potently) and adrenocorticotropic hormone (ACTH) (moderately). Ghrelin is also orexigenic, but its impact on substrate metabolism is controversial. We aimed to study direct effects of ghrelin on substrate metabolism and insulin sensitivity in human subjects.
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pubmed:commentsCorrections |
http://linkedlifedata.com/resource/pubmed/commentcorrection/18776138-10567011,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18776138-11061542,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18776138-11134161,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18776138-11739476,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18776138-13833973,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18776138-14742438,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18776138-15177917,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18776138-15328073,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18776138-15472202,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18776138-15522942,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18776138-15620416,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18776138-16314227,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18776138-16621911,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18776138-16679295,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18776138-17130496,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18776138-17311892,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18776138-18042651,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18776138-3278194,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18776138-6307793,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18776138-7030826,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18776138-7076741,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18776138-8904560,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18776138-9759505
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
AIM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Dec
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pubmed:issn |
1939-327X
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pubmed:author |
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pubmed:issnType |
Electronic
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pubmed:volume |
57
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
3205-10
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pubmed:dateRevised |
2010-9-21
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pubmed:meshHeading |
pubmed-meshheading:18776138-Adrenocorticotropic Hormone,
pubmed-meshheading:18776138-Cross-Over Studies,
pubmed-meshheading:18776138-Double-Blind Method,
pubmed-meshheading:18776138-Ghrelin,
pubmed-meshheading:18776138-Growth Hormone,
pubmed-meshheading:18776138-Humans,
pubmed-meshheading:18776138-Hydrocortisone,
pubmed-meshheading:18776138-Hypopituitarism,
pubmed-meshheading:18776138-Infusions, Intravenous,
pubmed-meshheading:18776138-Insulin Resistance,
pubmed-meshheading:18776138-Lipolysis,
pubmed-meshheading:18776138-Male,
pubmed-meshheading:18776138-Middle Aged,
pubmed-meshheading:18776138-Reference Values,
pubmed-meshheading:18776138-Signal Transduction,
pubmed-meshheading:18776138-Young Adult
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pubmed:year |
2008
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pubmed:articleTitle |
Ghrelin infusion in humans induces acute insulin resistance and lipolysis independent of growth hormone signaling.
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pubmed:affiliation |
Medical Department M (Endocrinology and Diabetes), Aarhus University Hospital, Aarhus, Denmark. etv@dadlnet.dk
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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