18725899

Source:http://linkedlifedata.com/resource/pubmed/id/18725899

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Predicate	Object
rdf:type	pubmed:Citation
lifeskim:mentions	umls-concept:C0026969, umls-concept:C0043240, umls-concept:C0374711, umls-concept:C1655731, umls-concept:C1705181, umls-concept:C2587213
pubmed:issue	9
pubmed:dateCreated	2009-1-22
pubmed:abstractText	Although the CNS has a robust innate ability to repair demyelinated axons, this capacity appears to dissipate with age. A study in this issue suggests that epigenetic processes participate in myelin repair and that the epigenetic response is less dynamic in older individuals.
pubmed:commentsCorrections	http://linkedlifedata.com/resource/pubmed/commentcorrection/18725899-19160500
pubmed:language	eng
pubmed:journal	http://linkedlifedata.com/resource/pubmed/journal/9809671
pubmed:citationSubset	IM
pubmed:status	MEDLINE
pubmed:month	Sep
pubmed:issn	1546-1726
pubmed:author	pubmed-author:PopkoBrianB
pubmed:issnType	Electronic
pubmed:volume	11
pubmed:owner	NLM
pubmed:authorsComplete	Y
pubmed:pagination	987-8
pubmed:meshHeading	pubmed-meshheading:18725899-Animals, pubmed-meshheading:18725899-Epigenesis, Genetic, pubmed-meshheading:18725899-Myelin Sheath, pubmed-meshheading:18725899-Nerve Regeneration
pubmed:year	2008
pubmed:articleTitle	Epigenetic control of myelin repair.
pubmed:affiliation	Jack Miller Center for Peripheral Neuropathy, Department of Neurology, University of Chicago, 5841 South Maryland Ave., MC2030, Chicago, Illinois 60637-1470, USA. bpopko@uchicago.edu
pubmed:publicationType	Journal Article, Comment, Review