Source:http://linkedlifedata.com/resource/pubmed/id/18722078
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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
1
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pubmed:dateCreated |
2008-12-22
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pubmed:abstractText |
Aggressive fibromatosis (syn. desmoid tumor) is a sporadically occurring neoplastic proliferation of fibroblasts originating from musculoaponeurotic planes, forming invasively growing masses without the capability to metastasize. The choice of treatment remains surgical resection with or without radiotherapy, and is characterized by high recurrence rates. Better understanding of the aetiology of aggressive fibromatosis is needed to be able to develop new treatment strategies to cope with the high recurrence rates.
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pubmed:language |
eng
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pubmed:journal | |
pubmed:citationSubset |
IM
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pubmed:chemical | |
pubmed:status |
MEDLINE
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pubmed:month |
Jan
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pubmed:issn |
1532-2157
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pubmed:author | |
pubmed:issnType |
Electronic
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pubmed:volume |
35
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
3-10
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pubmed:meshHeading |
pubmed-meshheading:18722078-Adenomatous Polyposis Coli Protein,
pubmed-meshheading:18722078-Fibromatosis, Aggressive,
pubmed-meshheading:18722078-Gene Expression Regulation, Neoplastic,
pubmed-meshheading:18722078-Genes, APC,
pubmed-meshheading:18722078-Humans,
pubmed-meshheading:18722078-Mutation,
pubmed-meshheading:18722078-Signal Transduction,
pubmed-meshheading:18722078-beta Catenin
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pubmed:year |
2009
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pubmed:articleTitle |
The role of APC and beta-catenin in the aetiology of aggressive fibromatosis (desmoid tumors).
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pubmed:affiliation |
Department of Surgery, Jeroen Bosch Hospital, Den Bosch, The Netherlands. d.lips@jbz.nl
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pubmed:publicationType |
Journal Article,
Review
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