Linked Life Data

18712277

Source:http://linkedlifedata.com/resource/pubmed/id/18712277

Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
18
pubmed:dateCreated
2008-9-24
pubmed:abstractText
Glutamate, by activation of metabotropic receptors (mGluRs), can lead to a reduction of synaptic efficacy at many synapses. These forms of synaptic plasticity are referred to as long-term depression (mGluR-LTD). We will distinguish between mGluR-LTD induced by pre- or postsynaptic receptors and mGluR-LTD induced by the locus of the expression mechanism of the synaptic depression. We will also review recent evidence that mGluR-mediated responses themselves are subject to depression, which may constitute a form of metaplasticity.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Sep
pubmed:issn
1420-682X
pubmed:author
pubmed:issnType
Print
pubmed:volume
65
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
2913-23
pubmed:meshHeading
pubmed:year
2008
pubmed:articleTitle
Mechanisms of synaptic depression triggered by metabotropic glutamate receptors.
pubmed:affiliation
Dept. of Basic Neurosciences, Medical Faculty, University of Geneva, 1211 Geneva, Switzerland.
pubmed:publicationType
Journal Article, Review