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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
10
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pubmed:dateCreated |
2008-10-9
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pubmed:abstractText |
Complement activation and ineffective clearance of complement-bearing immune complexes via erythrocytes contribute to the pathogenesis of systemic lupus erythematosus (SLE). Abnormally high levels of erythrocyte C4d and low levels of complement receptor 1 (CD35) have been reported in SLE and might have diagnostic utility. We investigated whether erythrocyte C4d and complement receptor 1 were specific for SLE and whether there was any association with disease activity.
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pubmed:grant |
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pubmed:language |
eng
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pubmed:journal |
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|
pubmed:citationSubset |
IM
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pubmed:chemical |
|
|
pubmed:status |
MEDLINE
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pubmed:month |
Oct
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pubmed:issn |
0315-162X
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:volume |
35
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
1989-93
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pubmed:meshHeading |
pubmed-meshheading:18709693-Biological Markers,
pubmed-meshheading:18709693-Case-Control Studies,
pubmed-meshheading:18709693-Complement C4b,
pubmed-meshheading:18709693-Erythrocytes,
pubmed-meshheading:18709693-Female,
pubmed-meshheading:18709693-Humans,
pubmed-meshheading:18709693-Lupus Erythematosus, Systemic,
pubmed-meshheading:18709693-Male,
pubmed-meshheading:18709693-Middle Aged,
pubmed-meshheading:18709693-Peptide Fragments,
pubmed-meshheading:18709693-Receptors, Complement 3b,
pubmed-meshheading:18709693-Severity of Illness Index
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pubmed:year |
2008
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pubmed:articleTitle |
Erythrocyte C4d and complement receptor 1 in systemic lupus erythematosus.
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pubmed:affiliation |
Mount Sinai Medical Center, New York, New York, USA.
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pubmed:publicationType |
Journal Article,
Research Support, N.I.H., Extramural
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