| pubmed-article:1870188 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:1870188 | lifeskim:mentions | umls-concept:C1000250 | lld:lifeskim |
| pubmed-article:1870188 | lifeskim:mentions | umls-concept:C0035668 | lld:lifeskim |
| pubmed-article:1870188 | lifeskim:mentions | umls-concept:C1519249 | lld:lifeskim |
| pubmed-article:1870188 | lifeskim:mentions | umls-concept:C0456389 | lld:lifeskim |
| pubmed-article:1870188 | lifeskim:mentions | umls-concept:C0392760 | lld:lifeskim |
| pubmed-article:1870188 | lifeskim:mentions | umls-concept:C1515655 | lld:lifeskim |
| pubmed-article:1870188 | lifeskim:mentions | umls-concept:C1515926 | lld:lifeskim |
| pubmed-article:1870188 | pubmed:issue | 9 | lld:pubmed |
| pubmed-article:1870188 | pubmed:dateCreated | 1991-9-19 | lld:pubmed |
| pubmed-article:1870188 | pubmed:abstractText | Turnip crinkle virus is one of several single-stranded RNA plant viruses associated with defective interfering (DI) RNAs. A complete cDNA copy of a 344-base DI RNA (DI RNA G) was cloned downstream from a T7 RNA polymerase promoter. Transcripts synthesized in vitro were infectious when inoculated with helper virus on turnip plants. Studies of the infectivity of DI transcripts containing deletions, insertions, and single-base changes suggest that (i) in general, only the 5' two-thirds of the molecule can tolerate mutations; (ii) between 52 and 67 bases of terminal 5' sequence are required for infectivity; (iii) nucleotides in positions 68 to 138 are not specifically involved in RNA infectivity; (iv) DI RNA G molecules smaller than 327 bases are not amplified efficiently in plants. | lld:pubmed |
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| pubmed-article:1870188 | pubmed:language | eng | lld:pubmed |
| pubmed-article:1870188 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:1870188 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:1870188 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:1870188 | pubmed:month | Sep | lld:pubmed |
| pubmed-article:1870188 | pubmed:issn | 0022-538X | lld:pubmed |
| pubmed-article:1870188 | pubmed:author | pubmed-author:LiX HXH | lld:pubmed |
| pubmed-article:1870188 | pubmed:author | pubmed-author:SimonA EAE | lld:pubmed |
| pubmed-article:1870188 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:1870188 | pubmed:volume | 65 | lld:pubmed |
| pubmed-article:1870188 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:1870188 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:1870188 | pubmed:pagination | 4582-90 | lld:pubmed |
| pubmed-article:1870188 | pubmed:dateRevised | 2010-9-9 | lld:pubmed |
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| pubmed-article:1870188 | pubmed:year | 1991 | lld:pubmed |
| pubmed-article:1870188 | pubmed:articleTitle | In vivo accumulation of a turnip crinkle virus defective interfering RNA is affected by alterations in size and sequence. | lld:pubmed |
| pubmed-article:1870188 | pubmed:affiliation | Department of Plant Pathology, University of Massachusetts, Amherst 01003. | lld:pubmed |
| pubmed-article:1870188 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:1870188 | pubmed:publicationType | Research Support, U.S. Gov't, Non-P.H.S. | lld:pubmed |
| pubmed-article:1870188 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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