| pubmed-article:18701807 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:18701807 | lifeskim:mentions | umls-concept:C0007634 | lld:lifeskim |
| pubmed-article:18701807 | lifeskim:mentions | umls-concept:C0035647 | lld:lifeskim |
| pubmed-article:18701807 | lifeskim:mentions | umls-concept:C0007589 | lld:lifeskim |
| pubmed-article:18701807 | lifeskim:mentions | umls-concept:C0053574 | lld:lifeskim |
| pubmed-article:18701807 | lifeskim:mentions | umls-concept:C0205245 | lld:lifeskim |
| pubmed-article:18701807 | lifeskim:mentions | umls-concept:C1704675 | lld:lifeskim |
| pubmed-article:18701807 | lifeskim:mentions | umls-concept:C2587213 | lld:lifeskim |
| pubmed-article:18701807 | lifeskim:mentions | umls-concept:C1511938 | lld:lifeskim |
| pubmed-article:18701807 | lifeskim:mentions | umls-concept:C1514485 | lld:lifeskim |
| pubmed-article:18701807 | pubmed:issue | 1-4 | lld:pubmed |
| pubmed-article:18701807 | pubmed:dateCreated | 2008-12-9 | lld:pubmed |
| pubmed-article:18701807 | pubmed:abstractText | Biglycan (BGN) and WISP-1 are 2 extracellular matrix proteins that bind to each other and colocalize in mineralizing tissue. Here we show that WISP-1 abrogates the repression of proliferation in bone marrow stromal cells induced by BGN. We also demonstrate that WISP-1 and its variant WISP-1va can alleviate the repressed osteogenic differentiation caused by the absence of BGN. These preliminary data suggest that WISP-1 and BGN may functionally interact and control each other's activity, thus regulating the differentiation and proliferation of osteogenic cells. | lld:pubmed |
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| pubmed-article:18701807 | pubmed:language | eng | lld:pubmed |
| pubmed-article:18701807 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:18701807 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:18701807 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:18701807 | pubmed:issn | 1422-6421 | lld:pubmed |
| pubmed-article:18701807 | pubmed:author | pubmed-author:YoungMarian... | lld:pubmed |
| pubmed-article:18701807 | pubmed:author | pubmed-author:FisherLarry... | lld:pubmed |
| pubmed-article:18701807 | pubmed:author | pubmed-author:BiYanmingY | lld:pubmed |
| pubmed-article:18701807 | pubmed:author | pubmed-author:KuznetsovSerg... | lld:pubmed |
| pubmed-article:18701807 | pubmed:author | pubmed-author:InksonColette... | lld:pubmed |
| pubmed-article:18701807 | pubmed:author | pubmed-author:OnoMitsuakiM | lld:pubmed |
| pubmed-article:18701807 | pubmed:copyrightInfo | Copyright 2008 S. Karger AG, Basel. | lld:pubmed |
| pubmed-article:18701807 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:18701807 | pubmed:volume | 189 | lld:pubmed |
| pubmed-article:18701807 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:18701807 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:18701807 | pubmed:pagination | 153-7 | lld:pubmed |
| pubmed-article:18701807 | pubmed:dateRevised | 2010-11-18 | lld:pubmed |
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| pubmed-article:18701807 | pubmed:year | 2009 | lld:pubmed |
| pubmed-article:18701807 | pubmed:articleTitle | The potential functional interaction of biglycan and WISP-1 in controlling differentiation and proliferation of osteogenic cells. | lld:pubmed |
| pubmed-article:18701807 | pubmed:affiliation | Craniofacial and Skeletal Diseases Branch, National Institute of Dental and Craniofacial Research, National Institutes of Health, DHHS Bethesda, Bethesda, Md. 20892-4320, USA. | lld:pubmed |
| pubmed-article:18701807 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:18701807 | pubmed:publicationType | Research Support, N.I.H., Intramural | lld:pubmed |
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