| pubmed-article:18688011 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:18688011 | lifeskim:mentions | umls-concept:C0035820 | lld:lifeskim |
| pubmed-article:18688011 | lifeskim:mentions | umls-concept:C0080093 | lld:lifeskim |
| pubmed-article:18688011 | lifeskim:mentions | umls-concept:C0475224 | lld:lifeskim |
| pubmed-article:18688011 | lifeskim:mentions | umls-concept:C1704410 | lld:lifeskim |
| pubmed-article:18688011 | lifeskim:mentions | umls-concept:C2754100 | lld:lifeskim |
| pubmed-article:18688011 | lifeskim:mentions | umls-concept:C0443199 | lld:lifeskim |
| pubmed-article:18688011 | lifeskim:mentions | umls-concept:C1152805 | lld:lifeskim |
| pubmed-article:18688011 | lifeskim:mentions | umls-concept:C0449560 | lld:lifeskim |
| pubmed-article:18688011 | pubmed:issue | 11 | lld:pubmed |
| pubmed-article:18688011 | pubmed:dateCreated | 2008-10-28 | lld:pubmed |
| pubmed-article:18688011 | pubmed:abstractText | Activation of NMDA subtypes of glutamate receptors is implicated in cell damage induced by ischemia as well as for the establishment of ischemic tolerance after ischemic preconditioning in animal models. We investigated the contributions of NR2A- and NR2B-containing NMDA receptors to ischemic cell death and ischemic tolerance in a rat model of transient global ischemia. | lld:pubmed |
| pubmed-article:18688011 | pubmed:language | eng | lld:pubmed |
| pubmed-article:18688011 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:18688011 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:18688011 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:18688011 | pubmed:month | Nov | lld:pubmed |
| pubmed-article:18688011 | pubmed:issn | 1524-4628 | lld:pubmed |
| pubmed-article:18688011 | pubmed:author | pubmed-author:LinN CNC | lld:pubmed |
| pubmed-article:18688011 | pubmed:author | pubmed-author:ChenQianQ | lld:pubmed |
| pubmed-article:18688011 | pubmed:author | pubmed-author:DannW JWJ | lld:pubmed |
| pubmed-article:18688011 | pubmed:author | pubmed-author:ZhouYangY | lld:pubmed |
| pubmed-article:18688011 | pubmed:author | pubmed-author:XiongZhi-QiZQ | lld:pubmed |
| pubmed-article:18688011 | pubmed:author | pubmed-author:DuanWen-HuWH | lld:pubmed |
| pubmed-article:18688011 | pubmed:author | pubmed-author:FengXiao-YanX... | lld:pubmed |
| pubmed-article:18688011 | pubmed:author | pubmed-author:LuTing-JiaTJ | lld:pubmed |
| pubmed-article:18688011 | pubmed:author | pubmed-author:ChenXiao-Jing... | lld:pubmed |
| pubmed-article:18688011 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:18688011 | pubmed:volume | 39 | lld:pubmed |
| pubmed-article:18688011 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:18688011 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:18688011 | pubmed:pagination | 3042-8 | lld:pubmed |
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| pubmed-article:18688011 | pubmed:year | 2008 | lld:pubmed |
| pubmed-article:18688011 | pubmed:articleTitle | Differential roles of NMDA receptor subtypes in ischemic neuronal cell death and ischemic tolerance. | lld:pubmed |
| pubmed-article:18688011 | pubmed:affiliation | Institute of Neuroscience, State Key Laboratory of Neuroscience, Shanghai, China. | lld:pubmed |
| pubmed-article:18688011 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:18688011 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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