| pubmed-article:18617779 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:18617779 | lifeskim:mentions | umls-concept:C0030705 | lld:lifeskim |
| pubmed-article:18617779 | lifeskim:mentions | umls-concept:C0012634 | lld:lifeskim |
| pubmed-article:18617779 | lifeskim:mentions | umls-concept:C0010346 | lld:lifeskim |
| pubmed-article:18617779 | lifeskim:mentions | umls-concept:C0004927 | lld:lifeskim |
| pubmed-article:18617779 | lifeskim:mentions | umls-concept:C1439296 | lld:lifeskim |
| pubmed-article:18617779 | lifeskim:mentions | umls-concept:C0332281 | lld:lifeskim |
| pubmed-article:18617779 | lifeskim:mentions | umls-concept:C1882417 | lld:lifeskim |
| pubmed-article:18617779 | pubmed:issue | 8 | lld:pubmed |
| pubmed-article:18617779 | pubmed:dateCreated | 2008-7-11 | lld:pubmed |
| pubmed-article:18617779 | pubmed:abstractText | CX3CR1, the receptor of CX3CL1/fractalkine, is involved in regulation of inflammatory response and the CX3CR1-I249-M280 naturally occurring mutants are associated with altered binding to the ligand. Our aim was to evaluate the frequency of CX3CR1 V249I and T280M polymorphisms and NOD2/CARD15 mutations in Crohn's disease patients and to search for a relationship with phenotype. | lld:pubmed |
| pubmed-article:18617779 | pubmed:language | eng | lld:pubmed |
| pubmed-article:18617779 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:18617779 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:18617779 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:18617779 | pubmed:month | Aug | lld:pubmed |
| pubmed-article:18617779 | pubmed:issn | 0954-691X | lld:pubmed |
| pubmed-article:18617779 | pubmed:author | pubmed-author:DeybachJean-C... | lld:pubmed |
| pubmed-article:18617779 | pubmed:author | pubmed-author:SobhaniIradjI | lld:pubmed |
| pubmed-article:18617779 | pubmed:author | pubmed-author:SouléJean-Cla... | lld:pubmed |
| pubmed-article:18617779 | pubmed:author | pubmed-author:CoffinBenoitB | lld:pubmed |
| pubmed-article:18617779 | pubmed:author | pubmed-author:JianRaymondR | lld:pubmed |
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| pubmed-article:18617779 | pubmed:author | pubmed-author:LamorilJérôme... | lld:pubmed |
| pubmed-article:18617779 | pubmed:author | pubmed-author:SabateJean-Ma... | lld:pubmed |
| pubmed-article:18617779 | pubmed:author | pubmed-author:JouetPaulineP | lld:pubmed |
| pubmed-article:18617779 | pubmed:author | pubmed-author:HarnoisFloren... | lld:pubmed |
| pubmed-article:18617779 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:18617779 | pubmed:volume | 20 | lld:pubmed |
| pubmed-article:18617779 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:18617779 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:18617779 | pubmed:pagination | 748-55 | lld:pubmed |
| pubmed-article:18617779 | pubmed:dateRevised | 2009-10-16 | lld:pubmed |
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| pubmed-article:18617779 | pubmed:year | 2008 | lld:pubmed |
| pubmed-article:18617779 | pubmed:articleTitle | The V249I polymorphism of the CX3CR1 gene is associated with fibrostenotic disease behavior in patients with Crohn's disease. | lld:pubmed |
| pubmed-article:18617779 | pubmed:affiliation | Department of Gastroenterology and Hepatology, AP-HP, Louis Mourier Hospital, Colombes, France. | lld:pubmed |
| pubmed-article:18617779 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:18617779 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| pubmed-article:18617779 | pubmed:publicationType | Multicenter Study | lld:pubmed |
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