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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
7
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pubmed:dateCreated |
2008-6-27
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pubmed:abstractText |
The mechanisms of interaction between Helicobacter pylori infection and low-dose aspirin in the induction of duodenal erosions are not fully understood. The aim of this study was to investigate the effects of low-dose aspirin on the induction of duodenal erosions, the expression of cyclooxygenases and prostaglandin (PG)-E(2) levels in healthy subjects according to their H. pylori status.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:issn |
1502-7708
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pubmed:author |
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pubmed:issnType |
Electronic
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pubmed:volume |
43
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
801-9
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pubmed:meshHeading |
pubmed-meshheading:18584518-Adult,
pubmed-meshheading:18584518-Anti-Inflammatory Agents, Non-Steroidal,
pubmed-meshheading:18584518-Aspirin,
pubmed-meshheading:18584518-Cyclooxygenase Inhibitors,
pubmed-meshheading:18584518-Dinoprostone,
pubmed-meshheading:18584518-Duodenal Ulcer,
pubmed-meshheading:18584518-Duodenum,
pubmed-meshheading:18584518-Female,
pubmed-meshheading:18584518-Helicobacter Infections,
pubmed-meshheading:18584518-Helicobacter pylori,
pubmed-meshheading:18584518-Humans,
pubmed-meshheading:18584518-Immunohistochemistry,
pubmed-meshheading:18584518-Intestinal Mucosa,
pubmed-meshheading:18584518-Male,
pubmed-meshheading:18584518-Prostaglandin-Endoperoxide Synthases
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pubmed:year |
2008
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pubmed:articleTitle |
Short-term/low-dose aspirin-induced duodenal erosions are not dependent on Helicobacter pylori infection, cyclooxygenase expression and prostaglandin E2 levels.
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pubmed:affiliation |
Department of Gastroenterology, Hepatology and Infectious Diseases, Otto-von-Guericke University, Magdeburg, Germany.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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