| pubmed-article:18571585 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:18571585 | lifeskim:mentions | umls-concept:C0346647 | lld:lifeskim |
| pubmed-article:18571585 | lifeskim:mentions | umls-concept:C0021753 | lld:lifeskim |
| pubmed-article:18571585 | lifeskim:mentions | umls-concept:C0334227 | lld:lifeskim |
| pubmed-article:18571585 | lifeskim:mentions | umls-concept:C0387583 | lld:lifeskim |
| pubmed-article:18571585 | lifeskim:mentions | umls-concept:C0041904 | lld:lifeskim |
| pubmed-article:18571585 | lifeskim:mentions | umls-concept:C1513475 | lld:lifeskim |
| pubmed-article:18571585 | pubmed:issue | 1 | lld:pubmed |
| pubmed-article:18571585 | pubmed:dateCreated | 2008-6-23 | lld:pubmed |
| pubmed-article:18571585 | pubmed:abstractText | Pancreatic cancer is a very aggressive malignancy and efficient therapeutic options are still largely lacking. The importance of interactions between tumor cells and surrounding stromal elements, eg, mononuclear cells, for chemoresistance have been increasingly recognized. In addition, cyclooxygenase-2 is thought to be an important mediator of chemoresistance in several malignancies. The aim of this study was to explore the role of mononuclear cells in pancreatic cancer chemoresistance. | lld:pubmed |
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| pubmed-article:18571585 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:18571585 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:18571585 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:18571585 | pubmed:month | Jul | lld:pubmed |
| pubmed-article:18571585 | pubmed:issn | 1532-7361 | lld:pubmed |
| pubmed-article:18571585 | pubmed:author | pubmed-author:EiblGuidoG | lld:pubmed |
| pubmed-article:18571585 | pubmed:author | pubmed-author:HinesOscar... | lld:pubmed |
| pubmed-article:18571585 | pubmed:author | pubmed-author:ReberHoward... | lld:pubmed |
| pubmed-article:18571585 | pubmed:author | pubmed-author:AngstElianeE | lld:pubmed |
| pubmed-article:18571585 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:18571585 | pubmed:volume | 144 | lld:pubmed |
| pubmed-article:18571585 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:18571585 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:18571585 | pubmed:pagination | 57-65 | lld:pubmed |
| pubmed-article:18571585 | pubmed:dateRevised | 2011-9-26 | lld:pubmed |
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| pubmed-article:18571585 | pubmed:meshHeading | pubmed-meshheading:18571585... | lld:pubmed |
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| pubmed-article:18571585 | pubmed:meshHeading | pubmed-meshheading:18571585... | lld:pubmed |
| pubmed-article:18571585 | pubmed:meshHeading | pubmed-meshheading:18571585... | lld:pubmed |
| pubmed-article:18571585 | pubmed:meshHeading | pubmed-meshheading:18571585... | lld:pubmed |
| pubmed-article:18571585 | pubmed:meshHeading | pubmed-meshheading:18571585... | lld:pubmed |
| pubmed-article:18571585 | pubmed:year | 2008 | lld:pubmed |
| pubmed-article:18571585 | pubmed:articleTitle | Mononuclear cell-derived interleukin-1 beta confers chemoresistance in pancreatic cancer cells by upregulation of cyclooxygenase-2. | lld:pubmed |
| pubmed-article:18571585 | pubmed:affiliation | Hirshberg Laboratories for Pancreatic Cancer Research, CURE, Digestive Diseases Research Center, David Geffen School of Medicine at UCLA, Los Angeles, California 90095, USA. | lld:pubmed |
| pubmed-article:18571585 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:18571585 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| pubmed-article:18571585 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
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