Source:http://linkedlifedata.com/resource/pubmed/id/18541994
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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
4
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pubmed:dateCreated |
2008-6-10
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pubmed:abstractText |
Over the past 18 months, the number of gene loci robustly associated with type 2 diabetes has risen from three to 18. In this study, we focus on explaining the genome-wide approach that has led to most of these discoveries and discuss some of the early insights the new gene loci have provided into the aetiology of type 2 diabetes.
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pubmed:language |
eng
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pubmed:journal | |
pubmed:citationSubset |
IM
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pubmed:chemical | |
pubmed:status |
MEDLINE
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pubmed:month |
Jul
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pubmed:issn |
1363-1950
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pubmed:author | |
pubmed:issnType |
Print
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pubmed:volume |
11
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
371-7
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pubmed:dateRevised |
2011-11-17
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pubmed:meshHeading |
pubmed-meshheading:18541994-Diabetes Mellitus, Type 2,
pubmed-meshheading:18541994-Genetic Predisposition to Disease,
pubmed-meshheading:18541994-Genetic Variation,
pubmed-meshheading:18541994-Genome, Human,
pubmed-meshheading:18541994-Humans,
pubmed-meshheading:18541994-Insulin,
pubmed-meshheading:18541994-Insulin-Secreting Cells
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pubmed:year |
2008
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pubmed:articleTitle |
New gene variants alter type 2 diabetes risk predominantly through reduced beta-cell function.
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pubmed:affiliation |
Genetics of Complex Traits, Institute of Biomedical and Clinical Science, Peninsula Medical School, Exeter, UK.
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pubmed:publicationType |
Journal Article,
Review
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