rdf:type |
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lifeskim:mentions |
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pubmed:issue |
3
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pubmed:dateCreated |
2008-7-24
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pubmed:abstractText |
Postischemic injury to the coronary vascular endothelium, in particular to the endothelial glycocalyx, may provoke fluid extravasation. Shedding of the glycocalyx is triggered by redox stress encountered during reperfusion and should be alleviated by the radical scavenger nitric oxide (NO). The objective of this study was to investigate the effect of exogenous administration of NO during reperfusion on both coronary endothelial glycocalyx and vascular integrity.
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pubmed:commentsCorrections |
http://linkedlifedata.com/resource/pubmed/commentcorrection/18518977-10604521,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18518977-10694618,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18518977-10747340,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18518977-11083232,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18518977-11300444,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18518977-12637366,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18518977-12960682,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18518977-1443194,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18518977-14659806,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18518977-14704229,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18518977-15114220,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18518977-1539722,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18518977-15964925,
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http://linkedlifedata.com/resource/pubmed/commentcorrection/18518977-16288777,
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http://linkedlifedata.com/resource/pubmed/commentcorrection/18518977-16732094,
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http://linkedlifedata.com/resource/pubmed/commentcorrection/18518977-9774189
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pubmed:language |
eng
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pubmed:journal |
|
pubmed:citationSubset |
IM
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pubmed:chemical |
|
pubmed:status |
MEDLINE
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pubmed:issn |
1466-609X
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pubmed:author |
|
pubmed:issnType |
Electronic
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pubmed:volume |
12
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
R73
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pubmed:dateRevised |
2009-11-18
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pubmed:meshHeading |
pubmed-meshheading:18518977-Adenine Nucleotides,
pubmed-meshheading:18518977-Animals,
pubmed-meshheading:18518977-Coronary Vessels,
pubmed-meshheading:18518977-Endothelium, Vascular,
pubmed-meshheading:18518977-Free Radical Scavengers,
pubmed-meshheading:18518977-Glycocalyx,
pubmed-meshheading:18518977-Guinea Pigs,
pubmed-meshheading:18518977-Heparin Lyase,
pubmed-meshheading:18518977-Heparitin Sulfate,
pubmed-meshheading:18518977-Lactic Acid,
pubmed-meshheading:18518977-Male,
pubmed-meshheading:18518977-Microscopy, Electron,
pubmed-meshheading:18518977-Myocardial Reperfusion,
pubmed-meshheading:18518977-Myocardial Reperfusion Injury,
pubmed-meshheading:18518977-Nitric Oxide,
pubmed-meshheading:18518977-Uric Acid
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pubmed:year |
2008
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pubmed:articleTitle |
Exogenous nitric oxide requires an endothelial glycocalyx to prevent postischemic coronary vascular leak in guinea pig hearts.
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pubmed:affiliation |
Clinic of Anesthesiology, Ludwig-Maximilians-University, Marchioninistrasse 15, 81377 Munich, Germany. dirk.bruegger@med.uni-muenchen.de
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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