18515308

Source:http://linkedlifedata.com/resource/pubmed/id/18515308

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Predicate	Object
rdf:type	pubmed:Citation
lifeskim:mentions	umls-concept:C0006674, umls-concept:C0018270, umls-concept:C0030518, umls-concept:C0087111, umls-concept:C0348016, umls-concept:C1292733, umls-concept:C1306673
pubmed:issue	11
pubmed:dateCreated	2008-10-16
pubmed:abstractText	Both the phenotypic alterations of parathyroid (PT) cells, e.g. down-regulation of the calcium-sensing receptor, and the increase of the PT cell number in nodular hyperplasia are the main causes of refractory secondary hyperparathyroidism. It is of great importance to prevent PT growth in an early stage.
pubmed:commentsCorrections	http://linkedlifedata.com/resource/pubmed/commentcorrection/18515308-10411707, http://linkedlifedata.com/resource/pubmed/commentcorrection/18515308-10620210, http://linkedlifedata.com/resource/pubmed/commentcorrection/18515308-12036954, http://linkedlifedata.com/resource/pubmed/commentcorrection/18515308-14520607, http://linkedlifedata.com/resource/pubmed/commentcorrection/18515308-15698459, http://linkedlifedata.com/resource/pubmed/commentcorrection/18515308-16395258, http://linkedlifedata.com/resource/pubmed/commentcorrection/18515308-16738533, http://linkedlifedata.com/resource/pubmed/commentcorrection/18515308-1739877, http://linkedlifedata.com/resource/pubmed/commentcorrection/18515308-17976085, http://linkedlifedata.com/resource/pubmed/commentcorrection/18515308-18790811, http://linkedlifedata.com/resource/pubmed/commentcorrection/18515308-2054233, http://linkedlifedata.com/resource/pubmed/commentcorrection/18515308-2370898, http://linkedlifedata.com/resource/pubmed/commentcorrection/18515308-2382648, http://linkedlifedata.com/resource/pubmed/commentcorrection/18515308-2631697, http://linkedlifedata.com/resource/pubmed/commentcorrection/18515308-2844214, http://linkedlifedata.com/resource/pubmed/commentcorrection/18515308-6941964, http://linkedlifedata.com/resource/pubmed/commentcorrection/18515308-7565429, http://linkedlifedata.com/resource/pubmed/commentcorrection/18515308-7816008, http://linkedlifedata.com/resource/pubmed/commentcorrection/18515308-7830860, http://linkedlifedata.com/resource/pubmed/commentcorrection/18515308-7861707, http://linkedlifedata.com/resource/pubmed/commentcorrection/18515308-7933819, http://linkedlifedata.com/resource/pubmed/commentcorrection/18515308-8261678, http://linkedlifedata.com/resource/pubmed/commentcorrection/18515308-8301927, http://linkedlifedata.com/resource/pubmed/commentcorrection/18515308-8397225, http://linkedlifedata.com/resource/pubmed/commentcorrection/18515308-8704116, http://linkedlifedata.com/resource/pubmed/commentcorrection/18515308-8780248, http://linkedlifedata.com/resource/pubmed/commentcorrection/18515308-8807569, http://linkedlifedata.com/resource/pubmed/commentcorrection/18515308-8823531, http://linkedlifedata.com/resource/pubmed/commentcorrection/18515308-8995751, http://linkedlifedata.com/resource/pubmed/commentcorrection/18515308-9058364, http://linkedlifedata.com/resource/pubmed/commentcorrection/18515308-9375818
pubmed:language	eng
pubmed:journal	http://linkedlifedata.com/resource/pubmed/journal/8706402
pubmed:citationSubset	IM
pubmed:chemical	http://linkedlifedata.com/resource/pubmed/chemical/Calcitriol, http://linkedlifedata.com/resource/pubmed/chemical/Calcium, http://linkedlifedata.com/resource/pubmed/chemical/Collagen Type I, http://linkedlifedata.com/resource/pubmed/chemical/Parathyroid Hormone, http://linkedlifedata.com/resource/pubmed/chemical/Peptides, http://linkedlifedata.com/resource/pubmed/chemical/Phosphorus, http://linkedlifedata.com/resource/pubmed/chemical/Vitamin D, http://linkedlifedata.com/resource/pubmed/chemical/collagen type I trimeric...
pubmed:status	MEDLINE
pubmed:month	Nov
pubmed:issn	1460-2385
pubmed:author	pubmed-author:HirakataHidekiH, pubmed-author:IidaMitsuoM, pubmed-author:TaniguchiMasatomoM, pubmed-author:TokumotoMasanoriM, pubmed-author:TsuruyaKazuhikoK
pubmed:issnType	Electronic
pubmed:volume	23
pubmed:owner	NLM
pubmed:authorsComplete	Y
pubmed:pagination	3662-9
pubmed:dateRevised	2010-11-18
pubmed:meshHeading	pubmed-meshheading:18515308-Administration, Oral, pubmed-meshheading:18515308-Aged, pubmed-meshheading:18515308-Bone and Bones, pubmed-meshheading:18515308-Calcitriol, pubmed-meshheading:18515308-Calcium, pubmed-meshheading:18515308-Collagen Type I, pubmed-meshheading:18515308-Dose-Response Relationship, Drug, pubmed-meshheading:18515308-Female, pubmed-meshheading:18515308-Humans, pubmed-meshheading:18515308-Hyperparathyroidism, Secondary, pubmed-meshheading:18515308-Hyperplasia, pubmed-meshheading:18515308-Injections, Intravenous, pubmed-meshheading:18515308-Longitudinal Studies, pubmed-meshheading:18515308-Male, pubmed-meshheading:18515308-Middle Aged, pubmed-meshheading:18515308-Parathyroid Glands, pubmed-meshheading:18515308-Parathyroid Hormone, pubmed-meshheading:18515308-Peptides, pubmed-meshheading:18515308-Phosphorus, pubmed-meshheading:18515308-Renal Dialysis, pubmed-meshheading:18515308-Renal Insufficiency, pubmed-meshheading:18515308-Vitamin D
pubmed:year	2008
pubmed:articleTitle	Intravenous calcitriol therapy in an early stage prevents parathyroid gland growth.
pubmed:affiliation	Department of Medicine and Clinical Science, Graduate School of Medical Sciences, Kyushu University, Higashi-Ku, Fukuoka 812-8582, Japan. masatomo@kcu.med.kyushu-u.ac.jp
pubmed:publicationType	Journal Article, Randomized Controlled Trial