| pubmed-article:18504144 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:18504144 | lifeskim:mentions | umls-concept:C0346647 | lld:lifeskim |
| pubmed-article:18504144 | lifeskim:mentions | umls-concept:C0042866 | lld:lifeskim |
| pubmed-article:18504144 | pubmed:issue | 2 | lld:pubmed |
| pubmed-article:18504144 | pubmed:dateCreated | 2009-2-2 | lld:pubmed |
| pubmed-article:18504144 | pubmed:abstractText | Sun exposure has been associated with lower death rates for pancreatic cancer in ecological studies. Skin exposure to solar ultraviolet B radiation induces cutaneous production of precursors to 25-hydroxy (OH) vitamin D (D) and is considered the primary contributor to vitamin D status in most populations. Pancreatic islet and duct cells express 25-(OH) D(3)-1alpha-hydroxylase that generates the biologically active 1,25-dihydroxy(OH)(2) D form. Thus, 25(OH)D concentrations could affect pancreatic function and possibly pancreatic cancer etiology. Serum 25-(OH)D is the major circulating vitamin D metabolite and is considered the best indicator of vitamin D status as determined by the sun and diet. Although recent prospective epidemiologic studies of higher predicted vitamin D status score and vitamin D intake and pancreatic cancer risk suggest protective associations, a nested case-control study showed a significant 3-fold increased risk for pancreatic cancer with higher vitamin D status. Limitations of these studies include the former do not measure vitamin D status on pancreatic cancer cases and the later was conducted in a male smoker population. More research is needed, particularly examination of pre-diagnostic vitamin D status and risk of pancreatic cancer, prior to conclusions for vitamin D's potential role in the etiology of this highly fatal cancer. | lld:pubmed |
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| pubmed-article:18504144 | pubmed:language | eng | lld:pubmed |
| pubmed-article:18504144 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:18504144 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:18504144 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:18504144 | pubmed:month | Feb | lld:pubmed |
| pubmed-article:18504144 | pubmed:issn | 1873-2585 | lld:pubmed |
| pubmed-article:18504144 | pubmed:author | pubmed-author:Stolzenberg-S... | lld:pubmed |
| pubmed-article:18504144 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:18504144 | pubmed:volume | 19 | lld:pubmed |
| pubmed-article:18504144 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:18504144 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:18504144 | pubmed:pagination | 89-95 | lld:pubmed |
| pubmed-article:18504144 | pubmed:dateRevised | 2011-9-28 | lld:pubmed |
| pubmed-article:18504144 | pubmed:meshHeading | pubmed-meshheading:18504144... | lld:pubmed |
| pubmed-article:18504144 | pubmed:meshHeading | pubmed-meshheading:18504144... | lld:pubmed |
| pubmed-article:18504144 | pubmed:meshHeading | pubmed-meshheading:18504144... | lld:pubmed |
| pubmed-article:18504144 | pubmed:meshHeading | pubmed-meshheading:18504144... | lld:pubmed |
| pubmed-article:18504144 | pubmed:year | 2009 | lld:pubmed |
| pubmed-article:18504144 | pubmed:articleTitle | Vitamin D and pancreatic cancer. | lld:pubmed |
| pubmed-article:18504144 | pubmed:affiliation | Nutritional Epidemiology Branch, Division of Cancer Epidemiology and Genetics, National Cancer Institute, National Institutes of Health, Department Health Human Services, Rockville, MD, USA. rs221z@nih.gov | lld:pubmed |
| pubmed-article:18504144 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:18504144 | pubmed:publicationType | Review | lld:pubmed |
| pubmed-article:18504144 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| pubmed-article:18504144 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
| pubmed-article:18504144 | pubmed:publicationType | Research Support, N.I.H., Intramural | lld:pubmed |
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