rdf:type |
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lifeskim:mentions |
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pubmed:issue |
1
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pubmed:dateCreated |
2008-6-16
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pubmed:abstractText |
Anorexia nervosa (AN) is a psychiatric illness that results in significant bone loss. Studies examining the neuroendocrine dysregulation that occurs in AN may increase understanding of endocrine systems that regulate bone mass. Peptide YY (PYY) is an anorexigenic peptide derived primarily from the intestine, with actions mediated via activation of Y receptors. We have previously shown that PYY levels are elevated in adolescents with AN. Y2 receptor knockout mice have increased bone mineral density (BMD) and thus PYY may play a role in regulating bone mass. We hypothesized that PYY levels would be inversely associated with BMD in women with AN.
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pubmed:grant |
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pubmed:commentsCorrections |
http://linkedlifedata.com/resource/pubmed/commentcorrection/18486583-10491228,
http://linkedlifedata.com/resource/pubmed/commentcorrection/18486583-10660043,
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Jul
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pubmed:issn |
8756-3282
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:volume |
43
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
135-9
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pubmed:dateRevised |
2011-10-19
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pubmed:meshHeading |
pubmed-meshheading:18486583-Absorptiometry, Photon,
pubmed-meshheading:18486583-Adult,
pubmed-meshheading:18486583-Animals,
pubmed-meshheading:18486583-Anorexia Nervosa,
pubmed-meshheading:18486583-Bone Density,
pubmed-meshheading:18486583-Cross-Sectional Studies,
pubmed-meshheading:18486583-Female,
pubmed-meshheading:18486583-Humans,
pubmed-meshheading:18486583-Mice,
pubmed-meshheading:18486583-Mice, Knockout,
pubmed-meshheading:18486583-Peptide YY
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pubmed:year |
2008
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pubmed:articleTitle |
Peptide YY (PYY) levels and bone mineral density (BMD) in women with anorexia nervosa.
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pubmed:affiliation |
Neuroendocrine Unit, Massachusetts General Hospital and Harvard Medical School, Boston, MA 02114, USA.
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pubmed:publicationType |
Journal Article
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