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rdf:type |
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lifeskim:mentions |
umls-concept:C0018270,
umls-concept:C0023810,
umls-concept:C0024623,
umls-concept:C0079488,
umls-concept:C0086418,
umls-concept:C0441655,
umls-concept:C0597357,
umls-concept:C0599946,
umls-concept:C0806987,
umls-concept:C1704259,
umls-concept:C1705987
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pubmed:issue |
10
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pubmed:dateCreated |
2008-5-16
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pubmed:abstractText |
Helicobacter pylori is reportedly involved in the development of gastric cancer. We investigated the mechanisms by which H. pylori affects gastric cancer growth and antitumor immunities in the host, focusing on H. pylori-derived lipopolysaccharide (LPS).
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pubmed:language |
eng
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pubmed:journal |
|
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pubmed:citationSubset |
IM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
May
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pubmed:issn |
1078-0432
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pubmed:author |
pubmed-author:ChochiKentaroK,
pubmed-author:IchikuraTakashiT,
pubmed-author:KawabataToshinobuT,
pubmed-author:KinoshitaManabuM,
pubmed-author:MajimaTakashiT,
pubmed-author:MochizukiHidetakaH,
pubmed-author:OnoSatoshiS,
pubmed-author:SekiShuhjiS,
pubmed-author:ShinomiyaNariyoshiN,
pubmed-author:SugasawaHidekazuH,
pubmed-author:TsujimotoHironoriH
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pubmed:issnType |
Print
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pubmed:day |
15
|
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pubmed:volume |
14
|
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pubmed:owner |
NLM
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|
pubmed:authorsComplete |
Y
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pubmed:pagination |
2909-17
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pubmed:meshHeading |
pubmed-meshheading:18483357-Cell Line, Tumor,
pubmed-meshheading:18483357-Cell Proliferation,
pubmed-meshheading:18483357-Cytotoxicity, Immunologic,
pubmed-meshheading:18483357-Helicobacter Infections,
pubmed-meshheading:18483357-Helicobacter pylori,
pubmed-meshheading:18483357-Humans,
pubmed-meshheading:18483357-Immunohistochemistry,
pubmed-meshheading:18483357-Leukocytes, Mononuclear,
pubmed-meshheading:18483357-Lipopolysaccharides,
pubmed-meshheading:18483357-Perforin,
pubmed-meshheading:18483357-Proliferating Cell Nuclear Antigen,
pubmed-meshheading:18483357-Reverse Transcriptase Polymerase Chain Reaction,
pubmed-meshheading:18483357-Stomach Neoplasms,
pubmed-meshheading:18483357-Toll-Like Receptor 4
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pubmed:year |
2008
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pubmed:articleTitle |
Helicobacter pylori augments growth of gastric cancers via the lipopolysaccharide-toll-like receptor 4 pathway whereas its lipopolysaccharide attenuates antitumor activities of human mononuclear cells.
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pubmed:affiliation |
Department of Surgery, National Defense Medical College, Namiki, Tokorozawa, Japan. grd1705@ndmc.ac.jp
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pubmed:publicationType |
Journal Article
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