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| pubmed-article:18473350 | lifeskim:mentions | umls-concept:C1299583 | lld:lifeskim |
| pubmed-article:18473350 | lifeskim:mentions | umls-concept:C1333903 | lld:lifeskim |
| pubmed-article:18473350 | lifeskim:mentions | umls-concept:C1514474 | lld:lifeskim |
| pubmed-article:18473350 | lifeskim:mentions | umls-concept:C0302350 | lld:lifeskim |
| pubmed-article:18473350 | lifeskim:mentions | umls-concept:C0679622 | lld:lifeskim |
| pubmed-article:18473350 | lifeskim:mentions | umls-concept:C0205314 | lld:lifeskim |
| pubmed-article:18473350 | lifeskim:mentions | umls-concept:C1608386 | lld:lifeskim |
| pubmed-article:18473350 | lifeskim:mentions | umls-concept:C1549571 | lld:lifeskim |
| pubmed-article:18473350 | lifeskim:mentions | umls-concept:C1521840 | lld:lifeskim |
| pubmed-article:18473350 | pubmed:issue | 2 | lld:pubmed |
| pubmed-article:18473350 | pubmed:dateCreated | 2008-7-15 | lld:pubmed |
| pubmed-article:18473350 | pubmed:abstractText | High mobility group AT-hook 1 (HMGA1) proteins are architectural transcription factors that are overexpressed by pancreatic adenocarcinomas. The authors hypothesized that tumor HMGA1 status represents a novel prognostic marker in pancreatic adenocarcinoma. They also tested the hypothesis that HMGA1 promotes anchorage-independent cellular proliferation and in vivo tumorigenicity. | lld:pubmed |
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| pubmed-article:18473350 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:18473350 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:18473350 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:18473350 | pubmed:month | Jul | lld:pubmed |
| pubmed-article:18473350 | pubmed:issn | 0008-543X | lld:pubmed |
| pubmed-article:18473350 | pubmed:author | pubmed-author:RedstonMarkM | lld:pubmed |
| pubmed-article:18473350 | pubmed:author | pubmed-author:RochaFlavioF | lld:pubmed |
| pubmed-article:18473350 | pubmed:author | pubmed-author:MatrosEvanE | lld:pubmed |
| pubmed-article:18473350 | pubmed:author | pubmed-author:LiauSiong-Sen... | lld:pubmed |
| pubmed-article:18473350 | pubmed:author | pubmed-author:WhangEdwardE | lld:pubmed |
| pubmed-article:18473350 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:18473350 | pubmed:day | 15 | lld:pubmed |
| pubmed-article:18473350 | pubmed:volume | 113 | lld:pubmed |
| pubmed-article:18473350 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:18473350 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:18473350 | pubmed:pagination | 302-14 | lld:pubmed |
| pubmed-article:18473350 | pubmed:dateRevised | 2011-3-1 | lld:pubmed |
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| pubmed-article:18473350 | pubmed:year | 2008 | lld:pubmed |
| pubmed-article:18473350 | pubmed:articleTitle | High mobility group AT-hook 1 (HMGA1) is an independent prognostic factor and novel therapeutic target in pancreatic adenocarcinoma. | lld:pubmed |
| pubmed-article:18473350 | pubmed:affiliation | Department of Surgery, Brigham and Women's Hospital, Harvard Medical School, Boston, Massachusetts 02115, USA. | lld:pubmed |
| pubmed-article:18473350 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:18473350 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| pubmed-article:18473350 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
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