| pubmed-article:18437348 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:18437348 | lifeskim:mentions | umls-concept:C0026809 | lld:lifeskim |
| pubmed-article:18437348 | lifeskim:mentions | umls-concept:C0014257 | lld:lifeskim |
| pubmed-article:18437348 | lifeskim:mentions | umls-concept:C0043096 | lld:lifeskim |
| pubmed-article:18437348 | lifeskim:mentions | umls-concept:C0015259 | lld:lifeskim |
| pubmed-article:18437348 | lifeskim:mentions | umls-concept:C0542341 | lld:lifeskim |
| pubmed-article:18437348 | lifeskim:mentions | umls-concept:C0449438 | lld:lifeskim |
| pubmed-article:18437348 | pubmed:issue | 7 | lld:pubmed |
| pubmed-article:18437348 | pubmed:dateCreated | 2008-6-3 | lld:pubmed |
| pubmed-article:18437348 | pubmed:abstractText | Exercise ameliorates oxidative stress-mediated diabetic vascular endothelial dysfunction through poorly defined mechanisms. We hypothesised that, in addition to improving metabolic parameters, upregulation of antioxidants such as superoxide dismutase (SOD) mediates exercise-induced reductions of oxidative stress and increased nitric oxide (NO) bioavailability, and also restores vasodilatation. | lld:pubmed |
| pubmed-article:18437348 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:18437348 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:18437348 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:18437348 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:18437348 | pubmed:month | Jul | lld:pubmed |
| pubmed-article:18437348 | pubmed:issn | 0012-186X | lld:pubmed |
| pubmed-article:18437348 | pubmed:author | pubmed-author:BrownseyR WRW | lld:pubmed |
| pubmed-article:18437348 | pubmed:author | pubmed-author:GhoshSS | lld:pubmed |
| pubmed-article:18437348 | pubmed:author | pubmed-author:RahmanM MMM | lld:pubmed |
| pubmed-article:18437348 | pubmed:author | pubmed-author:SallamNN | lld:pubmed |
| pubmed-article:18437348 | pubmed:author | pubmed-author:LaherII | lld:pubmed |
| pubmed-article:18437348 | pubmed:author | pubmed-author:KiefferT JTJ | lld:pubmed |
| pubmed-article:18437348 | pubmed:author | pubmed-author:KhazaeiMM | lld:pubmed |
| pubmed-article:18437348 | pubmed:author | pubmed-author:ElmiSS | lld:pubmed |
| pubmed-article:18437348 | pubmed:author | pubmed-author:Moien-Afshari... | lld:pubmed |
| pubmed-article:18437348 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:18437348 | pubmed:volume | 51 | lld:pubmed |
| pubmed-article:18437348 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:18437348 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:18437348 | pubmed:pagination | 1327-37 | lld:pubmed |
| pubmed-article:18437348 | pubmed:dateRevised | 2009-11-19 | lld:pubmed |
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| pubmed-article:18437348 | pubmed:year | 2008 | lld:pubmed |
| pubmed-article:18437348 | pubmed:articleTitle | Exercise restores endothelial function independently of weight loss or hyperglycaemic status in db/db mice. | lld:pubmed |
| pubmed-article:18437348 | pubmed:affiliation | Department of Pharmacology and Therapeutics, University of British Columbia, Vancouver, BC, V6T 1Z3, Canada. | lld:pubmed |
| pubmed-article:18437348 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:18437348 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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