pubmed-article:18437231 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:18437231 | lifeskim:mentions | umls-concept:C0035820 | lld:lifeskim |
pubmed-article:18437231 | lifeskim:mentions | umls-concept:C0011854 | lld:lifeskim |
pubmed-article:18437231 | lifeskim:mentions | umls-concept:C0026336 | lld:lifeskim |
pubmed-article:18437231 | lifeskim:mentions | umls-concept:C0006558 | lld:lifeskim |
pubmed-article:18437231 | lifeskim:mentions | umls-concept:C0349590 | lld:lifeskim |
pubmed-article:18437231 | pubmed:dateCreated | 2008-4-25 | lld:pubmed |
pubmed-article:18437231 | pubmed:abstractText | Patients with fulminant type 1 diabetes almost completely lack C-peptide even soon after the onset of the disease, and the deficiency continues for the rest of their life. Thus, fulminant type 1 diabetes could serve as a good model of nature to explore the physiological role of C-peptide. For example, patients with fulminant type 1 diabetes have diabetic chronic complications more frequently than those with classical autoimmune type 1 diabetes 5 years after the onset of diabetes, and the higher prevalence could be partly attributable to the complete lack of C-peptide in fulminant type 1 diabetes. | lld:pubmed |
pubmed-article:18437231 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:18437231 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:18437231 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:18437231 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:18437231 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:18437231 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:18437231 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:18437231 | pubmed:language | eng | lld:pubmed |
pubmed-article:18437231 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:18437231 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:18437231 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:18437231 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:18437231 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:18437231 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:18437231 | pubmed:issn | 1687-5303 | lld:pubmed |
pubmed-article:18437231 | pubmed:author | pubmed-author:ImagawaAA | lld:pubmed |
pubmed-article:18437231 | pubmed:author | pubmed-author:HanafusaToshi... | lld:pubmed |
pubmed-article:18437231 | pubmed:author | pubmed-author:Murase-Mishib... | lld:pubmed |
pubmed-article:18437231 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:18437231 | pubmed:volume | 2008 | lld:pubmed |
pubmed-article:18437231 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:18437231 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:18437231 | pubmed:pagination | 819123 | lld:pubmed |
pubmed-article:18437231 | pubmed:dateRevised | 2011-11-17 | lld:pubmed |
pubmed-article:18437231 | pubmed:meshHeading | pubmed-meshheading:18437231... | lld:pubmed |
pubmed-article:18437231 | pubmed:meshHeading | pubmed-meshheading:18437231... | lld:pubmed |
pubmed-article:18437231 | pubmed:meshHeading | pubmed-meshheading:18437231... | lld:pubmed |
pubmed-article:18437231 | pubmed:meshHeading | pubmed-meshheading:18437231... | lld:pubmed |
pubmed-article:18437231 | pubmed:meshHeading | pubmed-meshheading:18437231... | lld:pubmed |
pubmed-article:18437231 | pubmed:meshHeading | pubmed-meshheading:18437231... | lld:pubmed |
pubmed-article:18437231 | pubmed:meshHeading | pubmed-meshheading:18437231... | lld:pubmed |
pubmed-article:18437231 | pubmed:year | 2008 | lld:pubmed |
pubmed-article:18437231 | pubmed:articleTitle | Fulminant type 1 diabetes as a model of nature to explore the role of C-peptide. | lld:pubmed |
pubmed-article:18437231 | pubmed:affiliation | First Department of Internal Medicine, Osaka Medical College, 2-7 Daigaku-Machi, Takatsuki 569-8686, Japan. | lld:pubmed |
pubmed-article:18437231 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:18437231 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |