Source:http://linkedlifedata.com/resource/pubmed/id/18433785
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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
6
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pubmed:dateCreated |
2008-5-12
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pubmed:abstractText |
Nicotinic afferent pathways may be involved in the regulation of bladder inflammation. Based on that hypothesis we investigated the role of nicotinic signaling in a comparative analysis of 2 models of experimental bladder inflammation using protamine sulfate and cyclophosphamide.
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pubmed:grant | |
pubmed:language |
eng
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pubmed:journal | |
pubmed:citationSubset |
AIM
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pubmed:chemical | |
pubmed:status |
MEDLINE
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pubmed:month |
Jun
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pubmed:issn |
1527-3792
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pubmed:author | |
pubmed:issnType |
Electronic
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pubmed:volume |
179
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
2440-6
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pubmed:meshHeading |
pubmed-meshheading:18433785-Acute Disease,
pubmed-meshheading:18433785-Animals,
pubmed-meshheading:18433785-Cyclophosphamide,
pubmed-meshheading:18433785-Cystitis,
pubmed-meshheading:18433785-Disease Models, Animal,
pubmed-meshheading:18433785-Mice,
pubmed-meshheading:18433785-Mice, Inbred C57BL,
pubmed-meshheading:18433785-Protamines,
pubmed-meshheading:18433785-Receptors, Nicotinic
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pubmed:year |
2008
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pubmed:articleTitle |
Nicotinic signaling ameliorates acute bladder inflammation induced by protamine sulfate or cyclophosphamide.
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pubmed:affiliation |
Department of Urologic Surgery, Vanderbilt-Ingram Cancer Center, Vanderbilt University, Nashville, Tennessee 37232-2765, USA.
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pubmed:publicationType |
Journal Article,
Research Support, N.I.H., Extramural
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