| pubmed-article:18426833 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:18426833 | lifeskim:mentions | umls-concept:C0848558 | lld:lifeskim |
| pubmed-article:18426833 | lifeskim:mentions | umls-concept:C0017337 | lld:lifeskim |
| pubmed-article:18426833 | lifeskim:mentions | umls-concept:C1148798 | lld:lifeskim |
| pubmed-article:18426833 | lifeskim:mentions | umls-concept:C1314792 | lld:lifeskim |
| pubmed-article:18426833 | lifeskim:mentions | umls-concept:C1512498 | lld:lifeskim |
| pubmed-article:18426833 | pubmed:issue | 5 | lld:pubmed |
| pubmed-article:18426833 | pubmed:dateCreated | 2008-4-22 | lld:pubmed |
| pubmed-article:18426833 | pubmed:abstractText | Hypospadias is a common inborn error of the genital development, whose complex etiology remains elusive. Defects of the androgen metabolism and activity have been found in a subset of boys with hypospadias. Moreover, the balance between androgens and estrogens seems to be important to the proper male genital development. Activating transcription factor 3 (ATF3), an estrogen responsive gene, has been reported to be expressed during sexual development and up-regulated in hypospadic genital skin. We investigated ATF3 as a candidate gene for hypospadias. | lld:pubmed |
| pubmed-article:18426833 | pubmed:language | eng | lld:pubmed |
| pubmed-article:18426833 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:18426833 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:18426833 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:18426833 | pubmed:month | May | lld:pubmed |
| pubmed-article:18426833 | pubmed:issn | 1479-683X | lld:pubmed |
| pubmed-article:18426833 | pubmed:author | pubmed-author:TöhönenVirpiV | lld:pubmed |
| pubmed-article:18426833 | pubmed:author | pubmed-author:KockumIngridI | lld:pubmed |
| pubmed-article:18426833 | pubmed:author | pubmed-author:RadmayrChrist... | lld:pubmed |
| pubmed-article:18426833 | pubmed:author | pubmed-author:NordenskjöldA... | lld:pubmed |
| pubmed-article:18426833 | pubmed:author | pubmed-author:SchwentnerChr... | lld:pubmed |
| pubmed-article:18426833 | pubmed:author | pubmed-author:SöderhällCill... | lld:pubmed |
| pubmed-article:18426833 | pubmed:author | pubmed-author:Beleza-Meirel... | lld:pubmed |
| pubmed-article:18426833 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:18426833 | pubmed:volume | 158 | lld:pubmed |
| pubmed-article:18426833 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:18426833 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:18426833 | pubmed:pagination | 729-39 | lld:pubmed |
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| pubmed-article:18426833 | pubmed:meshHeading | pubmed-meshheading:18426833... | lld:pubmed |
| pubmed-article:18426833 | pubmed:year | 2008 | lld:pubmed |
| pubmed-article:18426833 | pubmed:articleTitle | Activating transcription factor 3: a hormone responsive gene in the etiology of hypospadias. | lld:pubmed |
| pubmed-article:18426833 | pubmed:affiliation | Department of Molecular Medicine and Surgery, Karolinska Institutet, SE-171 76 Stockholm, Sweden. | lld:pubmed |
| pubmed-article:18426833 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:18426833 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:18426833 | lld:pubmed |
