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| pubmed-article:18423809 | lifeskim:mentions | umls-concept:C0034656 | lld:lifeskim |
| pubmed-article:18423809 | lifeskim:mentions | umls-concept:C1611184 | lld:lifeskim |
| pubmed-article:18423809 | lifeskim:mentions | umls-concept:C0023779 | lld:lifeskim |
| pubmed-article:18423809 | lifeskim:mentions | umls-concept:C0717537 | lld:lifeskim |
| pubmed-article:18423809 | lifeskim:mentions | umls-concept:C0718050 | lld:lifeskim |
| pubmed-article:18423809 | lifeskim:mentions | umls-concept:C0449258 | lld:lifeskim |
| pubmed-article:18423809 | lifeskim:mentions | umls-concept:C2587213 | lld:lifeskim |
| pubmed-article:18423809 | lifeskim:mentions | umls-concept:C2603343 | lld:lifeskim |
| pubmed-article:18423809 | pubmed:issue | 6 | lld:pubmed |
| pubmed-article:18423809 | pubmed:dateCreated | 2008-5-26 | lld:pubmed |
| pubmed-article:18423809 | pubmed:databankReference | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:18423809 | pubmed:abstractText | Previous clinical trials showed that progression of coronary artery calcification (CAC) may be slower in hemodialysis patients treated with sevelamer than those treated with calcium-based phosphate binders. Because sevelamer decreases low-density lipoprotein cholesterol (LDL-C) levels, we hypothesized that intensive lowering of LDL-C levels with atorvastatin in hemodialysis patients treated with calcium acetate would result in CAC progression rates similar to those in sevelamer-treated patients. | lld:pubmed |
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| pubmed-article:18423809 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:18423809 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:18423809 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:18423809 | pubmed:month | Jun | lld:pubmed |
| pubmed-article:18423809 | pubmed:issn | 1523-6838 | lld:pubmed |
| pubmed-article:18423809 | pubmed:author | pubmed-author:XXX | lld:pubmed |
| pubmed-article:18423809 | pubmed:author | pubmed-author:KesslerPaul... | lld:pubmed |
| pubmed-article:18423809 | pubmed:author | pubmed-author:MuenzLarry... | lld:pubmed |
| pubmed-article:18423809 | pubmed:author | pubmed-author:HeDavid YDY | lld:pubmed |
| pubmed-article:18423809 | pubmed:author | pubmed-author:Diaz-BuxoJose... | lld:pubmed |
| pubmed-article:18423809 | pubmed:author | pubmed-author:QunibiWajehW | lld:pubmed |
| pubmed-article:18423809 | pubmed:author | pubmed-author:BudoffMathewM | lld:pubmed |
| pubmed-article:18423809 | pubmed:author | pubmed-author:CARE-2... | lld:pubmed |
| pubmed-article:18423809 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:18423809 | pubmed:volume | 51 | lld:pubmed |
| pubmed-article:18423809 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:18423809 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:18423809 | pubmed:pagination | 952-65 | lld:pubmed |
| pubmed-article:18423809 | pubmed:dateRevised | 2008-11-13 | lld:pubmed |
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| pubmed-article:18423809 | pubmed:year | 2008 | lld:pubmed |
| pubmed-article:18423809 | pubmed:articleTitle | A 1-year randomized trial of calcium acetate versus sevelamer on progression of coronary artery calcification in hemodialysis patients with comparable lipid control: the Calcium Acetate Renagel Evaluation-2 (CARE-2) study. | lld:pubmed |
| pubmed-article:18423809 | pubmed:affiliation | Department of Medicine, Division of Nephrology, University of Texas Health Sciences Center, San Antonio, TX 78229-3900, USA. qunibi@uthscsa.edu | lld:pubmed |
| pubmed-article:18423809 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:18423809 | pubmed:publicationType | Comparative Study | lld:pubmed |
| pubmed-article:18423809 | pubmed:publicationType | Randomized Controlled Trial | lld:pubmed |
| pubmed-article:18423809 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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