Source:http://linkedlifedata.com/resource/pubmed/id/18391785
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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
3
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pubmed:dateCreated |
2008-4-8
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pubmed:abstractText |
We discuss very recent studies that address the critical role of extracellular matrix in controlling the balance between vascular morphogenesis and regression. Much of this work suggests that a balance mechanism exists for controlling the extent of tissue vascularization involving downstream signaling events regulating endothelial cell behaviors in relation to their interactions with extracellular matrix molecules.
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pubmed:language |
eng
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pubmed:journal | |
pubmed:citationSubset |
IM
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pubmed:chemical | |
pubmed:status |
MEDLINE
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pubmed:month |
May
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pubmed:issn |
1065-6251
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pubmed:author | |
pubmed:issnType |
Print
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pubmed:volume |
15
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
197-203
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pubmed:meshHeading |
pubmed-meshheading:18391785-Cell Communication,
pubmed-meshheading:18391785-Collagen,
pubmed-meshheading:18391785-Extracellular Matrix,
pubmed-meshheading:18391785-Humans,
pubmed-meshheading:18391785-Matrix Metalloproteinases,
pubmed-meshheading:18391785-Neovascularization, Pathologic,
pubmed-meshheading:18391785-Neovascularization, Physiologic,
pubmed-meshheading:18391785-Signal Transduction
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pubmed:year |
2008
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pubmed:articleTitle |
Extracellular matrix mediates a molecular balance between vascular morphogenesis and regression.
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pubmed:affiliation |
Department of Medical Pharmacology and Physiology, University of Missouri School of Medicine, Columbia, Missouri 65212, USA. DavisGeo@missouri.edu
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pubmed:publicationType |
Journal Article,
Review
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