Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
6
pubmed:dateCreated
2008-5-28
pubmed:abstractText
Vulnerability of motor neurons in the spinal cord against ischemia is considered to play an important role in the development of delayed paraplegia after surgery of the thoracic aorta. However, the reasons for such vulnerability are not fully understood. Recently, the ubiquitin system has been reported to participate in neuronal cell death. In the present study, we investigated the expression of ubiquitin system molecules and discussed the relationship between the vulnerability and the ubiquitin system after transient ischemia in the spinal cord.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Jun
pubmed:issn
1524-4628
pubmed:author
pubmed:issnType
Electronic
pubmed:volume
39
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
1883-9
pubmed:meshHeading
pubmed:year
2008
pubmed:articleTitle
Ubiquitin-mediated stress response in the spinal cord after transient ischemia.
pubmed:affiliation
Department of Cardiovascular Surgery, Osaka University Graduate school of Medicine, 2-2 Yamadaoka, Suita, Osaka, 565-0871, Japan. yamataka@surg1.med.osaka-u.ac.jp
pubmed:publicationType
Journal Article