Source:http://linkedlifedata.com/resource/pubmed/id/18371398
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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
6
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pubmed:dateCreated |
2008-3-28
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pubmed:abstractText |
Tumor propagation by cancer stem cells (CSCs) requires their ability to self-renew, and yet the signal pathways involved in this process remain poorly defined. In the December issue of Cancer Cell, Zhao et al. (2007) provide compelling evidence that Wnt/beta-catenin signaling is crucial for the maintenance of chronic myelogenous leukemia (CML) stem cells.
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pubmed:language |
eng
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pubmed:journal | |
pubmed:citationSubset |
IM
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pubmed:chemical | |
pubmed:status |
MEDLINE
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pubmed:month |
Dec
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pubmed:issn |
1875-9777
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pubmed:author | |
pubmed:issnType |
Electronic
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pubmed:day |
13
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pubmed:volume |
1
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
597-8
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pubmed:dateRevised |
2009-5-20
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pubmed:meshHeading |
pubmed-meshheading:18371398-Disease Progression,
pubmed-meshheading:18371398-Gene Deletion,
pubmed-meshheading:18371398-Humans,
pubmed-meshheading:18371398-Leukemia,
pubmed-meshheading:18371398-Signal Transduction,
pubmed-meshheading:18371398-Stem Cells,
pubmed-meshheading:18371398-Wnt Proteins,
pubmed-meshheading:18371398-beta Catenin
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pubmed:year |
2007
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pubmed:articleTitle |
Knocking the Wnt out of the sails of leukemia stem cell development.
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pubmed:affiliation |
Department of Medicine III, Klinikum Grosshadern, D-81377 Munich, Germany.
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pubmed:publicationType |
Journal Article,
Review
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